Strangulated hernias in children. Postoperative ventral hernias Hernias of the white line of the abdomen

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Complications of abdominal hernias include: Strangulation Irreducibility Coprostasis Inflammation

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Strangulated hernia Strangulated hernia is the most common and dangerous complication requiring immediate surgical treatment. The organs that have entered the hernial sac are subject to compression (usually at the level of the neck of the hernial sac) in the hernial orifice.

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Pathological picture. In the strangulated organ, blood and lymph circulation are disrupted; due to venous stasis, fluid transudates into the intestinal wall, its lumen and the cavity of the hernial sac (hernial water). The intestine becomes cyanotic in color, the hernial water remains clear. Necrotic changes in the intestinal wall begin with the mucous membrane. The greatest damage occurs in the area of ​​the strangulation groove at the site of compression of the intestine by the pinching ring.

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Clinical picture and diagnosis. Clinical manifestations depend on the type of strangulation, the strangulated organ, and the time that has passed since the onset of the development of this complication. The main symptoms of a strangulated hernia are pain in the hernia area and irreducibility of a previously freely reducible hernia. The intensity of pain varies; sharp pain can cause shock. Local signs of strangulated hernia are sharp pain on palpation, compaction, and tension in the hernial protrusion. cough shock symptom is negative. During percussion, dullness is determined in cases where the hernial sac contains an omentum, bladder, and hernial water. If there is intestine containing gas in the hernial sac, then a tympanic percussion sound is determined.

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Treatment If a hernia is strangulated, emergency surgery is necessary. It is carried out in such a way as to open the hernial sac without cutting the strangulating ring and prevent the strangulated organs from slipping into the abdominal cavity. The operation is carried out in several stages.

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Stages of surgical treatment of a strangulated hernia: Layer-by-layer dissection of tissue up to the aponeurosis and exposure of the hernial sac. Opening the hernial sac, removing hernial water. Dissection of the pinching ring under visual control, so as not to damage the organs soldered to it from the inside. Determination of the viability of strangulated organs. Indisputable signs of intestinal non-viability are dark coloration, dull serous membrane, flabby wall, lack of pulsation of mesenteric vessels and intestinal peristalsis. Resection of a nonviable intestinal loop. Plastic surgery of hernial orifices.

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Irreducibility of a hernia The irreducibility of a hernia is caused by the presence in the hernial sac of fusions of internal organs with each other and with the hernial sac, formed as a result of their trauma and aseptic inflammation. Irreducibility can be partial, when one part of the hernia contents is reduced into the abdominal cavity, while the other remains irreducible. Long-term wearing of the bandage contributes to the development of irreducibility. Most often, umbilical, femoral and postoperative hernias are irreducible. Due to the development of multiple adhesions and chambers in the hernial sac, an irreducible hernia is often complicated by strangulation of organs in one of the chambers of the hernial sac or the development of adhesive intestinal obstruction.

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Coprostasis Stagnation of feces in the large intestine. This is a complication of a hernia in which the contents of the hernial sac are the large intestine. Coprostasis develops as a result of a disorder of intestinal motor function. Its development is facilitated by the irreducibility of the hernia, a sedentary lifestyle, and abundant food. Coprostasis is observed more often in obese patients of senile age, in men - with inguinal hernias, in women - with umbilical hernias. The main symptoms are persistent constipation, abdominal pain, nausea, and rarely vomiting. The hernial protrusion slowly increases as the colon fills with feces, it is almost painless, slightly tense, of a doughy consistency, the symptom of a cough impulse is positive. The general condition of the patients is of moderate severity.

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Treatment of coprostasis It is necessary to achieve the release of the colon from its contents. With reducible hernias, you should try to keep the hernia in a reduced state - in this case it is easier to restore intestinal motility. Small enemas with hypertonic sodium chloride solution, glycerin or repeated siphon enemas are used. The use of laxatives is contraindicated due to the risk of fecal impaction.
















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Presentation on the topic: Strangulated hernia

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A strangulated hernia is an acute compression of the contents of the hernial sac in the hernial orifice. This complication is observed, according to various statistics, in 8-20% of patients with abdominal hernias. Patients with strangulated hernias make up on average about 4-5% of all patients with “acute abdomen”. Strangulated hernias occur 1.5 times more often in women than in men. A strangulated hernia is usually accompanied by intestinal obstruction, which causes a fairly high mortality rate - 8-11%, and the mortality rate progressively increases with increasing time from the moment of strangulation to surgery.

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Strangulation without proper emergency medical care ultimately leads to necrosis of part or all of the organ. External hernias (located in defects of the pelvic floor or abdominal walls) and internal hernias (located in the walls of the diaphragm or in pockets of the peritoneum) can be subject to strangulation. Strangulated hernias come in different types. Whether the strangulated hernia belongs to one type or another depends on the anatomical region. The first classification of strangulated hernias is external and internal hernias.

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Clinical picture. Abdominal pain in the area of ​​the hernia is the main sign of strangulation; it usually occurs acutely due to physical stress, severe coughing, etc. Often the pain, although it occurs suddenly, intensifies gradually, and patients associate its occurrence with prolonged constipation. In some cases, it is so strong that patients cannot resist moaning and screaming. The pain is mostly constant. Particularly severe pain occurs when the intestinal mesentery is compressed in the hernial orifice, which leads to disruption of the blood supply to the strangulated intestinal loops.

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Types of strangulated hernias Strangulated hernias vary by anatomical region. First of all, they are divided into internal and external. Internal ones include diaphragmatic hernias. They are formed when part of the abdominal organs “enters” the chest cavity and is compressed by the muscles of the diaphragm. These hernias are rare and, as a rule, become strangulated even less often, since their symptoms appear very early. Most often, the following can be injured: the stomach, the lesser omentum (a connective tissue apron with a large amount of fatty tissue covering the stomach, liver and other organs of the upper abdomen) and the lower esophagus. All other hernias are external. Because they are based on the “exit” of part of the abdominal organs through the anterior abdominal wall. They, in turn, are: inguinal oblique and inguinal straight. This division is based on the direction of the hernial protrusion. ventral or hernia of the white line of the abdomen - the line that divides the abdomen into two halves.

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There are elastic and fecal strangulation. Elastic entrapment occurs suddenly, usually under the influence of some kind of stress (for example, lifting weights, heavy physical work). In this case, a larger than usual amount of viscera, such as loops of intestine, is pushed out of the abdominal cavity, which overwhelms the hernial sac. In a narrow place, in the hernial orifice, they are compressed, and thin-walled veins are compressed first, while blood continues to flow through the arteries for some time. Venous stagnation develops and fluid begins to leak from the veins into the cavity of the hernial sac; soon the intestinal wall becomes permeable to bacteria and the “hernial fluid” becomes infected. After some time, the intestinal loops become dead. In addition to the loop of intestine located in the hernial sac, the afferent loop of intestine is affected, which also loses its viability. Inflammation of the peritoneum develops, which causes death.

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Fecal strangulation develops slowly, gradually. It usually occurs with large inguinal hernias in patients suffering from constipation. The point here is that feces, filling the intestine located in the hernial sac, cannot overcome the obstacle - the bend of the efferent part of the intestine in the hernial orifice. Gradually, more loops of intestines are squeezed into the bag and the mesentery is compressed, just as with elastic strangulation. Therefore, this form of strangulation is very insidious, it can be expected with large hernias in elderly people suffering from constipation.

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In addition to the complete strangulation described above, with small ones, especially with femoral and umbilical hernias, so-called parietal strangulation can occur. This is an elastic strangulation, but in the hernial orifice it is not the mesentery of the intestinal loop that is strangulated, but a section of its wall. Diagnosing parietal entrapment is often very difficult. The passage of intestinal contents is not disrupted, and the hernial sac does not overflow. The patient complains only of more or less severe pain in the hernia area, and only with a very careful examination can sharp pain be detected at the site of the hernia orifice. Parietal strangulation is often the first sign of a femoral hernia. Slide description:

Another main complication of strangulated hernias is phlegmon of the hernial sac, which develops as a result of purulent melting of strangulated tissues. Then the clinical picture of a strangulated hernia is accompanied by signs of inflammation: fever, hyperemia of the skin under the hernial protrusion, etc. Stage of Grekov’s operation for a strangulated umbilical hernia, complicated by phlegmon of the hernial sac.

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Main complications First of all, the danger of a strangulated hernia lies in the appearance of necrosis (death) of the tissue that is subjected to compression. And also that part that is located outside the abdominal cavity. If, after some time, the hernia is strangulated (the reverse process of strangulation), a patch of dead tissue appears in the abdominal cavity. Toxins almost immediately begin to enter the blood. And quickly enough, immune cells “approach” the site of necrosis, causing aseptic inflammation. After a few hours, the intestinal flora penetrates beyond its boundaries and peritonitis develops. And this condition, even at the current stage of medical development, is characterized by high mortality.

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Treatment. Treatment of strangulated hernias is only surgical. The patient’s life directly depends on the time elapsed from the moment of injury to the operation. There are no contraindications for surgery for a strangulated hernia. If there is a suspicion of strangulation or self-reduction of the hernia, urgent hospitalization in the surgical department is necessary. In case of a serious condition of the patient with a full-blown picture of peritonitis or intestinal obstruction, infusion therapy during transportation is indicated.

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It is strictly forbidden to reduce a strangulated hernia, since this can very easily rupture the strangulated intestine. This will lead to the development of peritonitis. It happens that when shifting or transporting a patient, the strangulated hernia is independently reduced. Such patients still need to be hospitalized for observation, since often the reduced bowel loop is not viable.

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A cranial hernia is a rather rare developmental defect (occurs in 1 in 4000-8000 newborns), in which the membranes of the brain, and sometimes its substance, prolapse through defects in the bones of the skull (araphia, dysraphia).

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Etiology.

The occurrence of cranial hernias is associated with disruption of the development of the skull and brain in the early stages of the embryonic period, when the formation of the brain plate and its closure into the brain tube occur. Among the causes of cranial hernias are infectious and other diseases of the mother during pregnancy. Great importance is attached to heredity.

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Classification.

Anterior or sagittal (orbitonasal and frontal) 84.6%

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Posterior (occipital) 10.6%

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Hernias of the base of the skull (basal). 4.6%

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Most often there are anterior hernias, localized in places of embryonic fissures - at the root of the nose, at the inner edge of the orbit. Posterior cerebral hernias are located in the foramen magnum (above or below it). In the rarest basilar (basal) hernias, the defect is localized in the region of the anterior or middle cranial fossa, and the hernial contents protrude into the nasal or oral cavity.

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According to the contents of the hernial sac

Meningocele is a form in which the contents of the hernial sac are only the membranes of the brain (soft and arachnoid) and brain fluid. The dura mater and medulla remain intact. The dura mater, without participating in the formation of the hernial protrusion, is attached to the edges of the bone defect from the side of the cranial cavity.

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A true cerebral hernia, or meningoencephalocele, in which the sac contains the meninges and brain tissue.

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Encephalocystocele is the most severe form, in which the contents of the hernial sac are the medulla with part of the enlarged ventricle of the brain.

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The size of cerebral hernias varies greatly and can sometimes exceed the size of the child’s head. The size of the bone defect through which the hernia protrudes also varies, but most often does not exceed several centimeters. A hernia is not always covered by skin. This is an indication for urgent surgical treatment.

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Clinical picture.

When examining a child, a tumor-like formation is determined, most often located in the bridge of the nose, at the inner corner of the eye, or less often in the occipital region. The skin over the tumor is unchanged, palpation is painless. With anterior hernias, true hypertelorism attracts attention. The consistency of the hernial protrusion is soft-elastic, sometimes fluctuation is detected. When the child is restless, the formation becomes more intense, sometimes it is possible to detect fluctuations, which indicates a connection with the cranial cavity. The edges of a bone defect of the skull are determined much less frequently.

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With anterior cranial hernias, deformations of the facial skeleton, flattening of the bridge of the nose, widely spaced eyes, and strabismus come to the fore. Posterior cerebral hernias, in which the brain suffers more severely, are often accompanied by microcephaly and mental retardation. Symptoms of a central nervous system disorder may be absent, but if they are mild, especially in newborns, they are difficult to detect. At an older age, children are found to have lesions of the cranial nerves, asymmetry of tendon reflexes, and pathological reflexes; Sometimes paresis of the limbs and epileptic seizures are observed. Children with meningocele develop relatively normally, both physically and intellectually, while with a true cerebral hernia, pronounced disturbances in the central nervous system appear.

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Differential diagnosis.

Differential diagnosis of anterior cranial hernias is carried out mainly with dermoid cysts, which are sometimes located at the inner corner of the eye. Unlike cerebral hernias, dermoid cysts are usually small (rarely more than 1-1.5 cm) and have a dense consistency. The cause of the diagnostic error may be the usuration (the process of formation of a gap in the bone plate) of the bone plate, detected during radiographic examination and taken for a bone defect in a cerebral hernia

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Less often, a cranial hernia has to be differentiated from lipoma, hemangioma and lymphangioma. With tumor-like formations of soft tissues, the bone defect and pulsation characteristic of a cranial hernia are never determined.

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The following symptoms allow one to distinguish an intranasal cerebral hernia from a nasal polyp: a peculiar deformation of the nasal skeleton in the form of a wide bridge of the nose, bulging of one of its sides. The nasal septum is sharply pressed in the opposite direction. The color of the hernia is bluish in contrast to the gray color of the nasal polyp. Hernias are usually unilateral and have a wide base. When a hernia is punctured, cerebral fluid is detected in the punctate.

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Diagnostics

Analysis of complaints and medical history (questioning of parents): how the pregnancy with this child proceeded (did the pregnant woman have any infectious diseases, especially in the first trimester, did she take medications, drugs, alcohol, did she smoke); whether there were cases of such a developmental defect in the family. Neurological examination: assessment of the presence of a tumor-like soft formation on the head or face, the state of muscle tone (can be either increased or decreased), movements of the eyeballs (squint, limited mobility of the eyeballs may be observed). Examination by a pediatric otolaryngologist: examination of the nasal cavity, search for a hernia in the nose, assessment of the presence of leakage of cerebrospinal fluid (cerebrospinal fluid that provides nutrition and metabolism to the brain) from the nose. CT (computed tomography) and MRI (magnetic resonance imaging) of the head: allow you to study the structure of the brain layer by layer, assess the contents of the hernial sac (a protrusion, the walls of which are formed by the skin and membranes of the brain), and the size of the defect in the skull bones.

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Indications and contraindications for surgical treatment.

If there are no urgent vital indications (for example, in case of rapid growth of swelling and the danger of rupture of thinned tissues, it is necessary to operate in the first days after birth), then it is more profitable to operate on a hernia in children at the age of no earlier than 5-6 (2-3) years. . Surgical intervention is often resorted to in adults when patients decide to undergo surgery for cosmetic purposes. When a hernia is combined with dropsy of the brain, the dropsy is first cured and only after that the hernia is operated on. It is unacceptable to do the opposite, since after the defect is closed and the hernial sac is closed, the dropsy can sometimes progress or sharp headaches appear. Pleocytosis in the cerebrospinal fluid and the presence of meningeal symptoms are a contraindication for any method of surgical intervention.

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There are two methods of surgical intervention for anterior cerebral hernias:

extracranial (extracranial) - involves removing the hernial sac and closing the bone defect without opening the cranial cavity. It is used for laced hernias and small bone defects in children under 1 year of age.

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In 1881, N.V. Sklifosovsky for the first time undertook extracranial excision of a cerebral hernia, which resulted in recovery. Since that time, surgical thought has followed the path of radical surgery for congenital cerebral hernias.

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Depending on the size of the hernia and the condition of the skin, a linear, oval or bordering incision is made over it with the removal of excess skin. After this, the hernial sac is separated, isolated up to the external bone opening, and after stitching and tying the neck with a strong ligature, it is cut off. A small bone defect is closed by layer-by-layer suturing of soft tissue over it. If the diameter of the external opening of the hernial canal is more than 1 cm, it is closed either with a bone flap cut out from the outer plate of the skull bones, or by introducing an appropriate size organic glass pin into the bony hernial canal, after which the soft tissue is sutured in layers.

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However, frequent failures with this operation have led to the fact that in recent decades the extracranial method has begun to be used only for small hernias with a small bone defect, provided that the hernial sac is completely detached from the cerebrospinal fluid spaces of the brain. In cases where the hernial sac communicates with the cerebrospinal fluid spaces, the extracranial approach creates a risk of infection of the cranial cavity with the development of cerebrospinal fluid fistulas and meningitis.

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The intracranial method - closing the internal opening of a bone defect with an approach to it from the cranial cavity - is used in children over 1 year of age. The operation is performed in two stages: the first stage is intracranial plastic surgery of the skull bone defect, the second stage is removal of the hernial sac and rhinoplasty (performed after 3-6 months). The method was first proposed by P. A. Herzen (1926).

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A skin incision in the form of an arc is made along the border of the scalp of the forehead and both temples. A large aponeurotic skin flap is turned anteriorly towards the nose. The periosteum is cut along the edge of the bone incision, then along the sagittal suture and parallel to the upper edge of the orbits on both sides.

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Milling holes are placed along the cut line of the periosteum in such a way that after cutting through the bone, two bone flaps are formed, each of which could be turned to the side on a stem from the temporal muscle (in the form of two flaps). The anterior cut should be located as close as possible to the edge of the orbit, so that it is easier to approach the neck of the hernial sac. It is better to make a median cut of the bone, slightly retreating from the midline, so as not to damage the sagittal sinus. You should not be wary of damage to the frontal sinuses, since with anterior hernias they are rudimentary or completely absent.

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After exposing the dura mater, the latter is gradually separated from the bone with a wide spatula or rasp, pushing both frontal lobes posteriorly. Step by step, in this way, they approach the neck of the hernial sac and begin to separate it from the internal opening of the bony hernial canal using a curved rasp. Repression of the frontal lobes and isolation of the neck of the hernial sac is easier if 20-30 ml of cerebrospinal fluid are previously evacuated through the lumbar or ventricular route. The neck must be separated carefully so as not to damage the dura mater, which is very thin and can be soldered to the bone.

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After isolating the neck of the hernial sac, the dura mater in the area of ​​this neck is carefully dissected with a circular incision, 2-3 cm away from the bone defect, and then cut off. The dura mater on the side of the frontal lobe is carefully sutured with interrupted sutures. Then they begin to close the hernial bone hole from the side of the cranial cavity. A bone plate cut from the frontal bone or organic glass plastic is used.

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A month or later, the second stage of the operation is carried out to remove the hernial sac with its contents cut off during the first stage (as with the extracranial method). Sometimes the second stage turns out to be unnecessary since the hernial sac and its contents atrophy over time, scar and collapse.

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External abdominal hernia - protrusion of the internal organs of the abdominal cavity through defects in the muscular aponeurotic wall of the abdomen or pelvis along with the parietal layer of the peritoneum. An internal hernia is the penetration of abdominal organs into a normally existing or pathologically formed peritoneal sac (hernia of the omental bursa, diaphragmatic hernia, hernia of the duodenal-small intestinal fossa (Treits's fossa), retroduodenal fossa (mesenteric hernia) in the area of ​​peritoneal pockets near the cecum and sigmoid colon). The following concepts should be distinguished: Eventeration - the release of viscera from the abdominal cavity resulting from damage to the abdominal wall (wound, rupture). The parietal layer of the peritoneum is absent. Organ prolapse - the viscera protrude from any area and are not covered by a peritoneal layer (neither visceral nor parietal) For example: prolapse of the rectum, uterus, bladder.


They occur most often - up to 75% of all hernias - oblique and direct, congenital and acquired, unilateral and bilateral. Combinations with hydrocele of the spermatic cord, testicle, and cryptorchidism may occur. Up to 5% there are sliding hernias containing the cecum, bladder, kidney, ureter, uterus, sigmoid colon. If these organs are injured during surgery, the mortality rate reaches 27%.


Hernias that form in the vascular lacuna - the most common are typical (or medial) femoral hernias passing through the femoral canal, i.e. medial to the femoral vein. However, there may be so-called lateral vascular-lacunar femoral hernias, when the hernial protrusion is located outside the femoral artery, and - middle vascular-lacunar hernias, when the hernial sac is located under the pupart ligament above the femoral artery and vein. Hernias extending into the area of ​​the muscle lacuna are rare, located under the inguinal ligament in front of the iliopsoas muscle, lateral to the femoral artery. Hernias of the lacunar ligament are even less common, occurring in old women with a wide lacunar ligament.


Femoral hernias are more common in older women and are strangulated in 30-60%. In this case, compression of the hernial contents by the sharp edge of the lacunar ligament can quickly lead to necrosis. Therefore, femoral hernias require priority planned surgical treatment; as a rule, there is no need to dissect the hernial orifice, i.e. the probability of damage to “corona mortis” is minimal, occurring in 10-15% when a.obturatoria departs from a.epigastrica inferior or directly from a.ileaca externa.


Umbilical hernias account for 2-5% of all hernias. In 85% of cases they occur in women. The peculiarity of these hernias in children is that: they are detected in the first weeks of life (with incomplete fusion or weakness of the umbilical ring), or up to 3 years of age (against the background of constipation, coughing, crying), and are small in size (up to 1-2 cm) , are rarely infringed. Hernias of the white line of the abdomen occur in 90-95% of men. The most common are epigastric (82%), followed by mesogastric (~15%) and hypogastric in 0.3% of cases. The size of hernias of the white line is usually up to 10 cm, the contents are most often a strand of the greater omentum. The second most common group among abdominal hernias are postoperative hernias. Among them, it is necessary to highlight the group of postoperative hernias themselves - this is 3% of all “clean” laparotomies for any disease, 10% of festering surgical wounds and 10% of gunshot wounds of the abdominal cavity. It is also necessary to identify a group of recurrent hernias that occurred after a previous hernia repair. From 18 to 78%, depending on the type and location of the hernia, relapses occur. A special feature is a violation of the topographic-anatomical relationship of tissues, the presence of scar-altered structures of encapsulated “ligature granulomas”, “dormant infection” in scars, and sometimes the presence of fistulas.




Perineal hernias can be located anterior to the intersciatic line, starting from the vesico-uterine recess in women, and posterior to this line, starting from the vesico-rectal recess in men, and from the uterorectal recess in women from the pelvic peritoneum. Lateral abdominal hernias are formed along the Spigelian line and near the rectus abdominis muscles.


Occurs with a wide obturator canal. With a fully formed hernia, it is determined on the inner surface of the thigh under the adductor muscles. Most often, obturator hernias are incomplete, their clinical picture is unclear, they are recognized during surgery when they are strangulated.




Mandatory studies are studies that must be carried out for each patient: 1) clinical examination; 2) laboratory tests: CBC, TAM, blood group, Rh factor, blood clotting time, prothrombin index, glucose, blood urea; 3) ECG (in patients over 50 years of age or in patients who are planning surgery under general anesthesia); 4) consultation with a therapist (for the last group of patients); 5) fluorogram of the OGK during planned admission.


Recommended studies are studies whose significance has been proven when used for most patients: 1) electrolyte composition of the blood (Na+, K+, CL-); 2) blood biochemistry (billirubin, creatine); 3) coagulogram; 4) acid-base status and one-time blood composition; 5) cystoscopy (cystography) if there is a suspicion of the presence of a bladder in a hernia; 6) consultation with a urologist, breathing tests (for planned admission). Additional studies are studies that are significant for certain categories of patients, as well as those performed for the purpose of a more detailed assessment of homeostasis disorders. 1) plain radiography of the abdominal cavity; 2) Ultrasound of the abdominal cavity.


I. Conservative treatment is carried out only in 2 cases: in children under 2 years of age (application of tile-shaped bandages for umbilical hernias). Indications for surgery include rapid enlargement of the hernia, irreducibility and strangulation. in adult patients in the presence of contraindications to planned surgical treatment, i.e. conditions and diseases that make hernia repair life-threatening or are the cause of hernia formation (decompensated heart disease, heart failure, uncorrectable coagulopathy, active tuberculosis, malignant tumors and urethral strictures, prostate adenoma, pustular skin lesions, etc.) Such patients should be advised wearing a bandage, which gives only a symptomatic effect - it closes the hernial orifice and does not allow the hernial contents to escape into the hernial sac. It is impossible to recommend wearing a bandage to patients who have no contraindications to surgery, because Long-term wearing of a bandage contributes to the expansion of the hernial orifice (due to pressure on the tissue) and the formation of adhesions between the internal organs and the hernial sac.


4 hours), especially with extensive multi-chamber ventral hernia" title="Strangulated hernias are subject to emergency surgical treatment. There can be no contraindications to surgery. In patients in serious condition with many hours of strangulation of the hernial contents (> 4 hours), especially with extensive multi-chamber ventral hernia" class="link_thumb"> 14 !} Strangulated hernias are subject to emergency surgical treatment. There can be no contraindications to surgery. In patients in serious condition with long-term strangulation of the hernial contents (> 4 hours), especially with extensive multi-chamber ventral hernias, with clinical signs of endotoxemia and homeostasis disorders, as well as with a high anesthetic risk, it is possible to carry out emergency short-term preoperative infusion preparation in a volume of ml/kg the patient's body weight directly on the operating table. The main fundamental difference between the surgical procedure for a strangulated hernia (as opposed to an uncomplicated one) is the need for an initial opening of the hernial sac to fix and examine the strangulated organs and subsequent dissection of the strangulating ring. 4 hours), especially with extensive multi-chamber ventral hernias "> 4 hours), especially with extensive multi-chamber ventral hernias, with the presence of clinical signs of endotoxemia and homeostasis disorders, as well as with a high anesthetic risk, it is possible to carry out emergency short-term preoperative infusion preparation in the amount of 25-35 ml/kg of the patient's body weight directly on the operating table. The main fundamental difference between the surgical procedure for a strangulated hernia (as opposed to an uncomplicated hernia) is the need for an initial opening of the hernial sac for fixation and examination of the strangulated organs and subsequent dissection of the strangulated ring."> 4 hours), especially for extensive multi-chamber ventral hernia" title="Strangulated hernias are subject to emergency surgical treatment. There can be no contraindications to surgery. In patients in serious condition with strangulation of the hernial contents for many hours (> 4 hours), especially with extensive multi-chamber ventral hernia"> title="Strangulated hernias are subject to emergency surgical treatment. There can be no contraindications to surgery. In patients in serious condition with long-term strangulation of the hernial contents (> 4 hours), especially with extensive multi-chamber ventral hernias"> !}


In case of intestinal strangulation, the signs of its vitality are the following (must be reflected in the operation protocol): 1. restoration of the pink color of the serous cover; 2. restoration of peristalsis; 3. restoration of pulsation of mesenteric vessels. In doubtful cases, the recommended measure is the use of modern instrumental methods: laser Doppler flowmetry and intraoperative biomicroscopy. In the presence of intestinal necrosis, it is necessary to perform its resection and decompressive nasointestinal intubation in accordance with the standards adopted for the treatment of acute intestinal obstruction. If it is impossible to perform intestinal intubation in conditions of sharp overstretching of the intestinal loops from the herniolaparotomy access, the latter is either expanded (for p/o and umbilical hernias) or an additional median laparotomy access is made (for femoral, inguinal hernias).


In case of necrosis of the bladder wall, the strangulated part of the bladder is resected, an epicystostomy is applied, a urethral catheter is installed, and a flushing system is established. In case of strangulated Littre's hernia, Meckel's diverticulum is resected regardless of the state of its viability, using either a ligature-purse string method (similar to appendectomy) or a wedge resection of the intestine, including the base of the diverticulum. In case of necrosis of the wall of the cecum, it is necessary to perform a right hemicolectomy with ileotransverse anastomosis. If an error is made in the differential diagnosis and during surgery for a strangulated femoral hernia, not a hernia is discovered, but an enlarged inflamed lymph node - Pirogov, then it should not be removed due to the possibility of developing prolonged lymphorrhea and lymphostasis of the limb. The operation ends with the application of rare sutures to the wound and the provision of drainage to the inflamed lymph node.


Spontaneous reduction of a strangulated hernia If this reduction occurred before hospitalization or in the emergency room, then the patient must be hospitalized in the surgical department for diagnostic observation. In case of spontaneous reduction of the hernia, after prolonged strangulation (>4-6 hours), emergency laparoscopy is necessary. If, during observation for hours, the patient develops abdominal pain, positive symptoms of peritoneal irritation, increasing toxicosis, or clinical signs of intestinal obstruction, then it is necessary to perform a median laparotomy, revision and elimination of the cause of obstruction or resection of the strangulated organ according to indications. If spontaneous reduction of the hernia occurred during “induction of anesthesia” or the beginning of local anesthesia, then an opening of the hernial sac should be performed with inspection of nearby organs to identify the organ that has been strangulated and assess its viability. If it is difficult to find the strangulated organ, it is advisable to perform laparoscopy through the mouth of the hernial sac. If the condition of the abdominal cavity in the next 24 hours after the hernia repair does not cause concern, then the patient, after the necessary examination, can be operated on as planned. 4-6 hours), emergency laparoscopy is necessary. If, during observation within 24-48 hours, the patient develops abdominal pain, positive symptoms of peritoneal irritation, increasing toxicosis, or clinical signs of intestinal obstruction, then it is necessary to perform a median laparotomy, revision and elimination of the cause of obstruction or resection of the strangulated organ according to indications. If spontaneous reduction of the hernia occurred during “induction of anesthesia” or the beginning of local anesthesia, then an opening of the hernial sac should be performed with inspection of nearby organs to identify the organ that has been strangulated and assess its viability. If it is difficult to find the strangulated organ, it is advisable to perform laparoscopy through the mouth of the hernial sac. If the condition of the abdominal cavity in the next 24 hours after repair of the hernia does not cause concern, then the patient, after the necessary examination, can be operated on as planned.">


Phlegmon of the hernial sac The operation begins with a median laparotomy. If the intestinal loop is strangulated, then it is resected cm from the adductor and cm from the efferent section. The ends of the strangulated part of the intestine are ligated, and intestinal patency is restored by applying an “end to end” anastomosis and only if there is a significant discrepancy in the diameters of the intestine - “side to side”. At this stage of the operation, the peritoneal cavity must be isolated from the hernial sac cavity. To do this, the parietal peritoneum is dissected around the mouth of the sac and separated to the sides by 2 cm, the ends of the strangulated section of the intestine are re-ligated, cut off at the hernial orifice and the separated section of the parietal peritoneum is sutured over them. The median wound is sutured tightly in layers. Next, a herniotomy should be performed, the bottom of the hernial sac should be opened, then the strangulated ring should be cut so that the strangulated organ can be extracted and removed. The hernial sac is not isolated from the surrounding tissues. It is stitched at the cervix with tampons placed on it, which completes the intervention. Repair of the hernial orifice in conditions of purulent infection is strictly contraindicated, not only because it is doomed to failure in advance, but also because it can lead to the development of severe phlegmon of the abdominal wall. For phlegmon of the umbilical hernia, it is possible to use the circular through method of hernia repair according to Grekov.


The modern concept of hernia formation in relation to inguinal hernias in general has not been the subject of scientific debate for a long time. It is based on universally recognized work on the failure of the connective tissue structures of the posterior wall of the inguinal canal. It follows that the main pathogenetic principle of the treatment of inguinal hernias is to restore the posterior wall of the inguinal canal and give it the necessary mechanical strength, and hernioplasty methods should be assessed according to the main criterion: whether this goal of the operation is achieved or not. For more than 100 years, operations for inguinal hernias have been performed in Russia according to the principle founded by Bassini in 1884. Bassini’s operation was the basis for many other methods of hernioplasty.


Author Method of plastic surgery Year Recurrence rate Nesterenko Yu.A. Bassini, 2% Salov Yu.B. Kukudzhanova 19828.9% Mitasov I.G. Postemski 19856.0% Tran V. et al. Bassini, 0% Panos R. et al .Shouldice19927.0% Nyhus L.Nyhus19936.0%


The main reason for unsatisfactory treatment results with traditional methods of inguinal canal plastic surgery is the convergence of heterogeneous tissues under tension, which contradicts the basic principles of surgery. The muscles sutured to the inguinal ligament undergo degeneration, atrophy and cicatricial degeneration, and tension leads to ischemic necrosis of tissue along the suture line, their eruption and further relapse. The noted circumstances contributed to the development and improvement of a new treatment technology – tension-free hernioplasty. When repairing hernias of the white line of the abdomen, umbilical, small ventral postoperative hernias, in the absence of tissue tension, it is legal to use traditional surgical methods. In case of strangulated giant ventral hernias, which include most of the contents of the abdominal cavity, especially in elderly people with severe concomitant pathology, if it is impossible to perform tension-free hernioplasty due to the lack of an allograft, the hernial orifice should not be sutured, but only skin sutures should be placed on the wound.


The basis of tension-free repair is the elimination of the causes of relapses characteristic of traditional types of hernia repair: 1. Stitching of scar-modified tissues 2. Stitching of heterogeneous tissues (for inguinal hernias) 3. Tissue ischemia due to tension and compression by sutures With tension-free repair, the hernial orifice remains in its original state. The allomaterial covering the hernial orifice holds the tissues in a fixed position, stimulates the rapid formation of mature connective tissue, the strength of which is equal to or greater than the strength of the aponeurosis.


1. Elasticity so as not to cause bedsores of adjacent tissues 2. Resistance to infection 3. Do not cause pronounced inflammatory, allergic reactions 4. Porosity for the penetration of macrophages, fibroblasts, blood vessels and collagen fibers into the pores 5. Long-term mechanical strength and integrity 6. Not have carcinogenic properties


ABSORBABLE polygloctin-910 (vicryl) polyglycolic acid (Dexon) They are absorbed the day after surgery. The main manufacturers of mesh are "Ethicon", "B.Braun", "USSC USA", "Ekoflon". NON-ABSORBABLE polypropylene (Surgipro, Marlex, Prolene, Atrium) polyester (Mersilene) polytetrafluoroethylene (Tetlon, Gjre - Tex, Ecoflon) - maximally inert, can be placed intraperitoneally without the risk of causing an adhesive process, because due to the very low surface tension, it is difficult for the fibroplast to attach to the surface of this plate


It is used in 70-80% of cases for inguinal hernias in America and Europe. Its main principle is to strengthen the posterior wall of the inguinal canal with mesh allomaterial. Since Liechtenstein presented the results of more than 6,000 operations in 1989, this technique has been widely used throughout the world, with almost the same results as those achieved in the USA at the Liechtenstein Institute. This fact: learnability and reproducibility are the highest advantages of any technique.


Author Number of operations Age of patients Recurrence rate Lichtenstein L ,1% Horeyseck J ,25% Kux M ,9% Egiev V.N.,9% Friis M ,9% Advantages of hernioplasty according to Lichtenstein: 1. Significant reduction in the frequency of relapses (from 6 -14% to 1%): 2. Reduced surgical trauma, which reduces the severity of pain and allows patients to be discharged on days 3-4 3. Earlier return of patients to their normal lifestyle, due to the presence of an endoprosthesis in the wound, which provides additional strength


Suppuration of the postoperative wound (there is no need to remove the endoprosthesis) Rejection of the endoprosthesis Sensation of a foreign body. There are publications reporting the possibility of unpleasant sensations during coitus in men under 25 years of age, and therefore the use of this technique in this group of patients should be limited. Seroma Hematoma


General principles of implantation of endoprostheses: It is advisable to avoid contact of the allomaterial with the subcutaneous tissue to prevent the formation of seromas. After fixation to the tissues, the endoprosthesis should lie without tension. Contact of endoprostheses with the visceral peritoneum is unacceptable (except PTFE)


1. Preperitoneal implantation - the mesh is placed in the preperitoneal space, behind the aponeurosis, which eliminates the problem of seroma formation in the subcutaneous tissue, reduces the risk of infection and eliminates displacement of the prosthesis when intra-abdominal pressure increases.




3. Nadaponeurotic implantation - the mesh is located in the subcutaneous fat layer, fixed above the aponeurosis and muscles. With the “tension” option, plastic surgery of the hernial orifice is performed beforehand, and the mesh is placed over the sutures. With the “non-tension” method, the mesh is laid and fixed without tension to the aponeurosis around the perimeter using U-shaped sutures.


1. Laparoscopic preperitoneal prosthetic hernioplasty: In conditions of pneumoperitoneum, the peritoneum is dissected with scissors in a U-shape or arcuate manner, bending around the lateral and medial inguinal fossae from above. Next, the peritoneum is separated to the pubic bone. The hernial sac is bluntly separated from the elements of the spermatic cord and transverse fascia. To cover all three fossae (femoral, lateral and medial inguinal) - potential sites of hernia exit, the mesh dimensions must be at least 6 x 11 cm. The mesh can be applied without a cut (for direct inguinal hernias), or with a preliminary cut to wire it under elements of the spermatic cord. The prosthesis is fixed with a herniostapler using tantalum staples (10 pieces), avoiding damage to the iliac, lower epigastric, testicular vessels, vas deferens, and bladder.


Under endotracheal anesthesia, in conditions of tense carboxyperitoneum, the contents of the hernial sac are removed into the abdominal cavity; the sac itself may not be resected. A polytetrafluoroethylene implant is inserted into the abdominal cavity, exceeding the size of the hernia defect by at least 1 cm around the perimeter. The prosthesis is fixed over the peritoneum using a stapler with staples extended to 4.8 mm along the perimeter. IGeneral absolute contraindications are: pregnancy; concomitant diseases and conditions in which general anesthesia and planned surgical treatment are contraindicated. Relative contraindications include obesity of 3-4 degrees, adhesions in the abdominal cavity. If an adhesive process is suspected, the question of the possibility of surgery should be decided after performing diagnostic laparoscopy. The presence of additional special equipment allows operations to be performed under conditions of adhesions, however, the risk of developing intraoperative complications increases. II Local contraindications include: strangulated hernias; sliding; irreducible; giant inguinal hernias in combination with hydrocele of the spermatic cord and hydrocele of the testicular membranes. With increasing experience in performing endovideosurgical herneoplasty, it becomes possible to perform operations on patients with complicated forms of inguinal hernias.


They arise due to insufficient closure of the hernial orifice due to displacement of the implant when its fixation is not entirely reliable, as well as when the size of the prosthesis is insufficient. They usually occur within the first three weeks after surgery. In the later period, relapses are more rare, because The prosthesis, which has managed to grow with connective tissue, is securely fixed in the hernial orifice.

Donetsk National Medical University named after M. Gorky
Department of Faculty Surgery named after. K.T.Hovnatanyan
Assoc. Gredzhev F.A.
Donetsk 2008

Abdominal hernia (hernia abdominalis) is called

protrusion of internal organs covered with peritoneum
through natural or artificial abdominal openings
walls, pelvic floor, diaphragm under the outer integument
body or into another cavity.
The required components of a true hernia are:
1) hernial orifice; 2) hernial sac from the parietal
peritoneum; 3) hernial contents of the sac - organs
abdominal cavity.
Exit of internal organs through defects in
parietal peritoneum (i.e. not covered by peritoneum)
called prolapse (prolapse), or eventration.

Hernial orifice

natural or pathological opening in the muscularaponeurotic layer of the abdominal wall or fascial
sheath through which the hernial protrusion emerges.

Hernial sac

is part of the parietal peritoneum,
protruding through the hernial orifice. It distinguishes
the mouth is the initial part of the sac, the neck is the narrow section
sac located in the canal (in the thickness of the abdominal wall),
body - the largest part located outside
hernial orifice, and the bottom - the distal part of the sac.
The hernial sac can be single- or multi-cavity.

Hernial contents

internal organs located in the cavity of the hernial sac.
Any organ of the abdominal cavity can be in the hernial sac.
Most often it contains well-moving organs: large
omentum, small intestine, sigmoid colon, appendix. Hernial
contents can be easily reduced into the abdominal cavity (reducible
hernias), only partially reduced, not reduced (irreducible hernias)
or be strangulated in the hernial orifice (strangulated hernia).
It is especially important to distinguish strangulated hernias from irreducible ones, since
strangulation threatens the development of acute intestinal obstruction,
necrosis and gangrene of the intestine, peritonitis. If most of the internal
organs remain in the hernial sac for a long time, then
such hernias are called giant hernias (hernia pennagna). They have difficulty
reduced during surgery due to volume reduction
abdominal cavity and loss of space previously occupied by them.

External abdominal hernia

External abdominal hernias occur in 3-4% of total
population. Based on their origin, they distinguish between congenital and
acquired hernias. The latter are divided into force hernias
(due to a sharp increase in intra-abdominal pressure),
hernias from weakness due to muscle atrophy, reduction
tone and elasticity of the abdominal wall (in the elderly and
weakened persons). In addition, there are
postoperative and traumatic hernias. IN
Depending on the anatomical location, they are distinguished
hernias are inguinal, femoral, umbilical, lumbar, sciatic, obturator, perineal.

Internal abdominal hernias












intestines right and left.







Etiology and pathogenesis

Hernias most often occur in children under the age of 1
of the year. The number of patients gradually decreases to 10 years
age, after that it increases again until the age of 30-40
reaches its maximum. In old and senile age
An increase in the number of patients with hernias was also noted.
Inguinal hernias form most often (75%),
femoral (8%), umbilical (4%), and
postoperative (12%). All other forms of hernia
make up about 1%. Men are more likely to have inguinal
hernias, in women - femoral and umbilical.

Predisposing factors

Predisposing factors include heredity,
age (for example, weak abdominal wall in children of the first
years of life, atrophy of abdominal wall tissue in old
people), gender (features of the structure of the pelvis and large sizes
femoral ring in women, weakness of the groin area and
formation of the inguinal canal in men), degree
fatness (rapid weight loss), abdominal wall injury,
postoperative scars, nerve palsy,
innervating the abdominal wall. These factors
contribute to weakening of the abdominal wall.

Producing factors

Producing factors cause an increase
intra-abdominal pressure; these include heavy
physical labor, difficult childbirth, difficulty
urination, constipation, prolonged cough. An effort,
contributing to an increase in intra-abdominal pressure,
may be single and sudden (heavy lifting)
or frequently recurring (cough).

The cause of a congenital hernia is
underdevelopment of the abdominal wall in the prenatal period:
embryonic umbilical hernia, fetal hernia
(hernia of the umbilical cord), non-union of the vaginal
process of the peritoneum. First, hernias form
gate and hernial sac, later as a result of physical
efforts internal organs penetrate into the hernial sac.
For acquired hernias, the hernial sac and internal
organs exit through the internal opening of the canal, then
through the external (femoral canal, inguinal canal).

(general principles)

The main symptoms of the disease are protrusion and pain in the hernia area
when straining, coughing, physical stress, walking, when the patient is in an upright position.
The protrusion disappears or decreases in a horizontal position or after manual
reduction.
The protrusion gradually increases and takes on an oval or round shape. For hernias,
acutely arising at the moment of a sharp increase in intra-abdominal pressure, patients feel
severe pain in the area of ​​the forming hernia, sudden appearance of protrusion of the abdominal wall
and in rare cases, hemorrhage into surrounding tissue.
The patient is examined in a vertical and horizontal position. View in vertical
position makes it possible to determine, when straining and coughing, protrusions that were previously invisible, and when
For large hernias, their largest size is determined. When percussing a hernial protrusion
a tympanic sound is detected if there is intestine containing gases in the hernial sac, and
dullness of percussion sound, if there is a greater omentum or organ in the bag, not
containing gas.
During palpation, the consistency of the hernial contents is determined (elastic consistency
has an intestinal loop, a lobed structure of soft consistency - the greater omentum).
With the patient in a horizontal position, the reducibility of the contents of the hernial sac is determined. IN
At the moment of reduction of a large hernia, a characteristic rumbling of the intestines can be heard.
After reducing the hernial contents with a finger inserted into the hernial orifice, the
size, shape of the external opening of the hernial orifice. When a patient coughs, a finger
the examiner feels the tremors of the protruding peritoneum and adjacent organs - a symptom
cough impulse; it is characteristic of an external abdominal hernia.
For large hernias, to determine the nature of the hernial contents,
X-ray examination of the digestive tract, bladder.

Treatment (general principles)

Conservative treatment is carried out for umbilical hernias in children. It lies in
the use of dressings with pelota, which prevents the release of internal organs. U
adults use various types of bandages. Wearing a bandage is prescribed
patients who cannot be operated on due to the presence of serious
contraindications to surgery (chronic diseases of the heart, lungs, kidneys in
stages of decompensation, liver cirrhosis, dermatitis, eczema, malignant
neoplasms). Wearing a bandage prevents internal organs from coming out
into the hernial sac and promotes temporary closure of the hernial orifice.
The use of a bandage is possible only for reducible hernias. Long lasting it
wearing can lead to atrophy of the abdominal wall tissue, the formation of adhesions
between the internal organs and the hernial sac, i.e. to the development of irreducible
hernias
Surgical treatment is the main method of preventing such severe
complications of a hernia, such as strangulation, inflammation, etc.
For uncomplicated hernias, the tissue above the hernial protrusion is dissected,
hernial orifice, isolate the hernial sac and open it. Set
the contents of the bag into the abdominal cavity, stitch and bandage the neck
hernial sac. The sac is cut off and the abdominal wall is strengthened in the area of ​​the hernia
gate by plastic surgery with local tissues, less often with alloplastic materials.
Hernia repair is performed under local or general anesthesia.
Prevention. Preventing the development of hernias in children consists of following
hygiene of infants: proper care of the navel, rational feeding,
regulation of intestinal function. Adults need regular exercise
physical culture and sports to strengthen both muscles and the body in
in general.
Early identification of persons suffering from abdominal hernias is of great importance and
performing the operation before complications develop. For this you need
preventive examinations of the population, in particular schoolchildren and the elderly
age.

Inguinal hernia

Inguinal hernias account for 75% of all hernias. Among the patients
with inguinal hernias the share of men is 90-97%.
Inguinal hernias can be congenital or acquired.

Embryological information

From the third month of intrauterine development of the male embryo
sex, the process of descent of the testicles begins. In area
a bulge develops in the internal inguinal ring
parietal peritoneum - processus vaginalis
peritoneum. In the subsequent months of intrauterine
development, further protrusion of the diverticulum occurs
peritoneum into the inguinal canal. By the end of the seventh month the testicles
begin to descend into the scrotum. By the time of birth
the child's testicles are located in the scrotum, the processus vaginalis
the peritoneum is overgrown. When it fails to heal, it forms
congenital inguinal hernia. In case of incomplete fusion
vaginal process of the peritoneum in certain areas
hydrocele of the spermatic cord (funicocele) occurs.

Anatomy of the groin area

When examining the anterior abdominal wall from the inside with
five folds can be seen on the sides of the abdominal cavity
peritoneum and depressions (pits), which are places
release of hernias. The external inguinal fossa is
internal opening of the inguinal canal, it is projected
approximately above the middle of the inguinal (pupart) ligament on
1.0-1.5 cm above it. Normally, the inguinal canal is
slit-like space filled with sperm in men
cord, in women - round ligament of the uterus. Inguinal
the canal runs obliquely at an angle to the inguinal ligament and
men has a length of 4.0-4.5 cm.

Inguinal canal and inguinal space

The walls of the inguinal canal are formed: Anterior - by the aponeurosis of the external oblique
abdominal muscles, lower - inguinal ligament, posterior - transverse fascia
abdomen, upper - free edges of the internal oblique and transverse muscles
belly.
The external (superficial) opening of the inguinal canal is formed by the legs
aponeurosis of the external oblique abdominal muscle, one of them is attached to
to the pubic tubercle, the other to the pubic fusion. External hole size
inguinal canal is different. Its transverse diameter is 1.2-3.0 cm,
longitudinal - 2.3-3.0 cm. In women, the external opening of the inguinal canal
slightly less than in men.
The internal oblique and transverse abdominal muscles, located in the groove
inguinal ligament, approach the spermatic cord and are thrown over it,
forming an inguinal space of different shapes and sizes. Boundaries of the inguinal
gap: below - the inguinal ligament, above - the edges of the internal oblique and
transverse abdominal muscles, on the medial side - the outer edge of the straight
abdominal muscles. The inguinal space may have a slit-like,
spindle-shaped or triangular in shape. Triangular groin shape
gap indicates weakness in the groin area.
At the site of the internal opening of the inguinal canal, the transverse fascia
it bends funnel-shaped and passes onto the spermatic cord, forming a common
the tunica vaginalis of the spermatic cord and testicle.
Round ligament of the uterus at the level of the external opening of the inguinal canal
is divided into fibers, some of which end on the pubic bone, the other
lost in the subcutaneous fatty tissue of the pubic area.

Congenital inguinal hernia

If the processus vaginalis of the peritoneum remains completely
unfused, then its cavity communicates freely with
peritoneal cavity. Subsequently, it is formed
congenital inguinal hernia, in which the vaginal
the process is a hernial sac. Congenital inguinal
hernias make up the bulk of hernias in children (90%).
However, adults also have congenital inguinal hernias.
(about 10-12%).

Acquired inguinal hernias

Acquired inguinal hernia. There are oblique
inguinal hernia and direct. Indirect inguinal hernia
passes through the external inguinal fossa, straight -
through the medial With a channel form, the bottom
the hernial sac reaches the external opening
inguinal canal. With a cord form of hernia
exits through the external opening of the inguinal canal and
located at different heights of the spermatic cord.
In the inguinal-scrotal form, the hernia descends into
scrotum, stretching it.

Sliding inguinal hernia

are formed when one of the walls of the hernial
the sac is an organ partially covered by peritoneum,
for example, bladder, cecum. Rarely hernial
the sac is absent, and the entire protrusion is formed only
those segments of the slipped organ that are not
covered with peritoneum.
Sliding hernias account for 1.0-1.5% of all inguinal hernias
hernia They arise due to mechanical contraction
peritoneum of the hernial sac of adjacent segments
intestines or bladder, devoid of serous cover.
It is necessary to know the anatomical features of the sliding
hernia, so as not to be opened during the operation instead
hernial sac, intestinal wall or bladder wall.

Clinical picture and diagnosis of inguinal hernias

It is not difficult to recognize a formed inguinal hernia. Typical is
medical history: sudden onset of a hernia during physical exertion
or the gradual development of a hernial protrusion, the appearance of a protrusion with
straining, in a vertical position of the patient's body and reduction - in
horizontal. Patients are concerned about pain in the hernia area, in the abdomen, feeling
discomfort when walking.
Examining the patient in an upright position gives an idea of ​​asymmetry
groin areas. If there is a protrusion of the abdominal wall, you can
determine its size and shape. Digital examination of the external orifice
inguinal canal is performed in a horizontal position of the patient after
reduction of the contents of the hernial sac. Doctor with index finger
invaginating the skin of the scrotum, enters the superficial opening of the inguinal
canal, located medially and slightly higher from the pubic tubercle. Fine
The superficial opening of the inguinal canal in men allows the tip of the finger to pass through.
When the posterior wall of the inguinal canal is weakened, you can freely insert the tip
finger behind the horizontal branch of the pubic bone, which cannot be done with
well-defined posterior wall formed by the transverse fascia of the abdomen.

It is mandatory to examine the scrotal organs (palpation of the seminal
cords, testes and epididymis).

Examination of the patient

Examination of the patient in an upright position gives an idea of
asymmetry of the groin areas. If there is a protrusion of the abdominal
walls, you can determine its size and shape.
Digital examination of the external opening of the inguinal canal
performed with the patient in a horizontal position after reduction
contents of the hernial sac. Doctor with index finger
invaginating the skin of the scrotum, enters the superficial opening
inguinal canal, located medially and slightly above the pubic canal
tubercle Normally, the superficial opening of the inguinal canal in men
misses the tip of the finger. When the posterior wall of the inguinal region is weakened
canal, you can freely place your fingertip behind the horizontal branch
pubic bone, which cannot be done with a well-defined posterior
wall formed by the transverse fascia of the abdomen.
Determine the symptom of cough impulse. Both inguinal canals are examined.
It is mandatory to examine the scrotal organs (palpation
spermatic cords, testes and epididymis).

Examination of the patient

Diagnosis of inguinal hernias in women is based on
inspection and palpation, since inserting a finger into the external
the opening of the inguinal canal is almost impossible.
In women, an inguinal hernia is differentiated from a cyst
round ligament of the uterus located in the inguinal canal. IN
Unlike a hernia, it does not change its size when
horizontal position of the patient, percussion sound above
it is always dull, and tympanitis is possible over the hernia.

Differential diagnosis

An inguinal hernia should be differentiated from hydrocele, varicocele, and
femoral hernia.
A hydrocele has a round or oval rather than pear-shaped shape, a densely elastic consistency, and a smooth surface. Palpable formation
cannot be distinguished from the testicle and its epididymis. Large hydrocele,
reaching the external opening of the inguinal canal, can be clearly separated from it
upon palpation. The percussion sound above the hydrocele is dull, above the hernia it may be
tympanic. An important method of differential diagnosis is
diaphanoscopy (transillumination). It is produced in a dark room using
a flashlight placed tightly on the surface of the scrotum. If palpable
If the formation contains a clear liquid, it will appear when transilluminated
have a reddish color. Intestinal loops located in the hernial sac,
the seal does not allow light rays to pass through.
Varicocele (varicose veins of the spermatic cord), in which
When the patient is in an upright position, dull arching pain appears in the
scrotum and there is a slight increase in its size. By palpation you can
detect serpentine dilatation of the veins of the spermatic cord. Dilated veins
They easily fall off when you press on them or when you lift the scrotum upward.
It should be borne in mind that varicocele can occur with (testicular pressure
veins with a tumor of the lower pole of the kidney.

Treatment

The main method is surgical treatment.
The main purpose of the operation is plastic surgery of the inguinal canal.
The operation is carried out in stages. First stage -
formation of access to the inguinal canal: in the inguinal
areas make an oblique incision parallel and above
inguinal ligament from the anterosuperior iliac spine
to the symphysis; dissect the aponeurosis of the external oblique muscle
belly; its upper flap is separated from the internal oblique and
transverse muscle, lower - from the spermatic cord,
exposing the groove of the inguinal ligament to the pubic
tubercle
The second stage is to isolate and remove the hernial sac;
In the third stage, the deep inguinal ring is sutured until
normal sizes (diameter 0.6-0.8 cm)
The fourth stage is the actual plastic surgery of the inguinal canal.

Access for inguinal hernia

When choosing a method of inguinal canal plastic surgery, you should
take into account that the main reason for the formation of inguinal
hernia is a weakness of its posterior wall.
For direct hernias and complex forms of inguinal hernias
(oblique with straightened canal, sliding, recurrent)
plastic surgery of the posterior wall of the inguinal
channel.
Strengthening its anterior wall with mandatory suturing
deep ring to normal size can be
used in children and young men with small
indirect inguinal hernias.

Stages of hernia repair

Stages of hernia repair

Stages of hernia repair

Stages of hernia repair

Stages of hernia repair

Stages of hernia repair

Stages of hernia repair

Stages of hernia repair

Stages of hernia repair

Stages of hernia repair

Methods of plastic surgery of the inguinal canal

The Bobrov-Girard method ensures strengthening of the anterior wall of the inguinal canal. Above
with the spermatic cord, the edges of the internal oblique and transverse are first sewn to the inguinal ligament
abdominal muscles, and then with separate sutures - the upper flap of the aponeurosis of the external oblique muscle
belly. The lower aponeurosis flap is fixed with sutures on the upper aponeurosis flap, thus forming
thus duplicating the aponeurosis of the external oblique abdominal muscle.
The Bassini method ensures strengthening of the posterior wall of the inguinal canal. After removal
of the hernial sac, the spermatic cord is moved to the side and an internal oblique is sutured under it
and the transverse muscle along with the transversus abdominis fascia to the inguinal ligament. Spermatic cord
placed on the formed muscle wall. Deep sutures help
restoration of the weakened posterior wall of the inguinal canal. Edges of the aponeurosis external oblique
the abdominal muscles stitch edge to edge above the spermatic cord.
Postempsky’s method involves complete elimination of the inguinal canal, inguinal space and
creating the inguinal canal with a completely new direction. Edge of the rectus sheath
the abdomen, together with the connected tendon of the internal oblique and transverse muscles, is sutured to
superior pubic ligament. Next, the upper aponeurosis flap along with the internal oblique and transverse
abdominal muscles are sutured to the pubiliac cord and to the inguinal ligament. These seams should
push the spermatic cord to the lateral side as far as possible. Lower flap of external aponeurosis
oblique abdominal muscle, passed under the spermatic cord, fixed over the upper flap
aponeurosis. The newly formed “inguinal canal” with the spermatic cord must pass through
the muscular aponeurotic layer in an oblique direction from back to front and from inside to outside so that
its inner and outer holes were not opposite each other. Spermatic cord
placed on the aponeurosis and the subcutaneous fatty tissue and skin are sutured over it.

Femoral hernia

Femoral hernias are located on the thigh in the area
femoral triangle and account for 5-8% of all
abdominal hernia.
Femoral hernias occur especially often in
women, which is explained by the greater severity
muscular and vascular lacunae and smaller
strength of the inguinal ligament.

Anatomy of femoral hernias

Between the inguinal ligament and the pelvic bones is located
space that is divided by the iliopectineal
fascia into two gaps - muscular and vascular. IN
the muscle lacunae contains the iliopsoas muscle and
femoral nerve. The vascular lacuna contains the femoral
artery with femoral vein.
Between the femoral vein and the lacunar ligament there is
space filled with fibrous connective tissue
and the Pirogov-Rosenmüller lymph node. This
the gap is called the femoral ring, through which
femoral hernia comes out.
Borders of the femoral ring: above - inguinal ligament; below -
ridge of pubic bone; outside - femoral vein; To
in the middle is the lacunar (gimbernate) ligament.
Under normal conditions, the femoral canal does not exist. He
formed during the formation of a femoral hernia. Oval
the fossa on the lata fascia of the thigh is the external opening
femoral canal.

Clinical picture and diagnosis

A characteristic sign of a femoral hernia is
protrusion in the area of ​​the femoral-inguinal flexion in
in the form of a small hemispherical formation
size located under the inguinal ligament
medially from the femoral vessels. Rarely hernial
the protrusion rises upward and is located
above the inguinal ligament.

Differential diagnosis

Femoral hernia is differentiated from inguinal hernia
hernias For a femoral irreducible hernia there may be
lipomas located in the upper
section of the femoral triangle. Lipoma has
lobular structure, not connected with the external
opening of the femoral canal. Simulate
femoral hernia can be enlarged lymphatic
nodes in the femoral triangle
(chronic lymphadenitis, tumor metastases to the lymph nodes).

Treatment of femoral hernias

Bassini method: an incision is made parallel to the inguinal ligament and
below it above the hernial protrusion. Hernial orifice
closed by suturing the inguinal and superior pubic ligaments.
Apply 3-4 stitches. The second row of seams between
the falciform edge of the fascia lata and the pectineal fascia
The femoral canal is sutured with fascia.
Ruggi's method - Parlavecchio: an incision is made as in the inguinal
hernia. The aponeurosis of the external oblique abdominal muscle is opened.
The inguinal space is exposed. Transversalis fascia is incised
in the longitudinal direction. Pushing back the preperitoneal
fiber, secrete the neck of the hernial sac. Hernial sac
removed from the femoral canal, opened, sutured and
deleted. The hernial orifice is closed by stitching
internal oblique, transverse muscle, superior edge
transverse fascia with superior pubic and inguinal ligaments.
Plastic surgery of the anterior wall of the inguinal canal is performed by
duplication of the aponeurosis of the external oblique muscle of the abdomen.

Umbilical hernias

An umbilical hernia is a protrusion of organs
abdominal cavity through an abdominal wall defect in
navel area. Highest incidence
observed among young children and people in
aged about 40 years. In women, umbilical hernia
occurs twice as often as in men, which is associated with
stretching of the umbilical ring during
pregnancy.

Treatment of umbilical hernias

Surgical only - autoplasty of the abdominal wall
Sapezhko or Mayo method.
Sapezhko method: with separate sutures, grabbing from one
on one side the edge of the aponeurosis of the white line of the abdomen, and on the other
sides - posteromedial part of the rectus sheath
abdomen, create a duplicate of the muscular aponeurotic
flaps in the longitudinal direction. At the same time, the flap
located superficially, hemmed to the bottom in the form
duplications.
Mayo method: two transverse incisions are used to excise the skin
along with the navel. After isolation and excision of the hernial
the hernial orifice expands in the transverse direction
two incisions through the linea alba and the anterior wall
sheaths of the rectus abdominis muscles to their inner edges.
The lower flap of the aponeurosis is sutured with U-shaped sutures under
upper, which is in the form of duplication with separate seams
sutured to the bottom flap.

Access for umbilical hernias

Sapezhko method

Mayo method

Hernia of the white line of the abdomen

Hernias of the white line of the abdomen can be
supra-umbilical, peri-umbilical and sub-umbilical.
The latter are extremely rare.
Peri-umbilical hernias are most often located on the side of the
navel
Characterized by the presence of pain in the epigastric region,
worsening after eating, with increased
intra-abdominal pressure. During examination
the patient is found to have typical hernia
symptoms. Research needs to be done to
identifying diseases accompanied by pain
in the epigastric region.

Treatment of hernias of the white line of the abdomen

The operation consists of closing the hole in
aponeurosis with a purse-string suture or separate
interrupted seams. With concomitant hernia
the divergence of the rectus abdominis muscle is used
Napalkov's method - dissect the rectus vagina
abdominal muscles along the inner edge and stitched
first the inner and then the outer edges of the leaves
dissected vaginas. In this way they create
doubling of the linea alba.

Rare types of abdominal hernias

Hernias of the xiphoid process are formed when there is a defect in it. Through
the openings in the xiphoid process may protrude like a preperitoneal
lipoma and true hernias. The diagnosis is made based on the detected
compaction in the area of ​​the xiphoid process, the presence of a defect in it and data
radiography of the xiphoid process.
A lateral hernia (lunar line hernia) emerges through a defect in that part
aponeurosis of the abdominal wall, which is located between the semilunar
(Spigelian) line (the border between the muscle and tendon parts
transverse abdominis muscle) and the outer edge of the rectus muscle. The hernia goes away
through the aponeuroses of the transverse and internal oblique abdominal muscles and
located under the aponeurosis of the external oblique abdominal muscle in the form
interstitial hernia (between the muscles of the abdominal wall). Often
complicated by infringement. Diagnosis is difficult and must be differentiated from
tumors and diseases of internal organs.
Lumbar hernias are rare. Their exit points are the upper and
lower lumbar triangles between the XII rib and the iliac crest
bones along the lateral edge of the latissimus dorsi (m. latissimus dorsi).
Hernias can be congenital or acquired; prone to infringement. Their
should be differentiated from abscesses and tumors.

Rare types of abdominal hernias

Obturator hernia (obturator foramen hernia) comes out along with
neurovascular bundle (vasa obturatoria, n. obturatorius) through the obturator
hole under the pectineus muscle (m. pectineus) and appears on the inner
surface of the upper thigh. More common in older women
due to weakening of the pelvic floor muscles. The hernia is usually small
size, can easily be mistaken for a femoral hernia.
Perineal hernias (anterior and posterior). Anterior perineal hernia
exits through the vesicouterine recess (excavaflo vesicouterina) of the peritoneum
into the labia majora in its central part. Posterior perineal hernia
exits through the rectouterine cavity (excavatfo rectouterina),
passes posteriorly from the intersciatic line through the fissures in the levator muscle
the anus, and exits into the subcutaneous fatty tissue, is located
in front or behind the anus. Perineal hernias are more common
observed in women. The contents of the hernial sac are urinary
bladder, genitals. Anterior perineal hernia in women
must be differentiated from an inguinal hernia, which also extends into
labia majora. Helps diagnose digital examination through
vagina; hernial protrusion of the perineal hernia can be palpated
between the vagina and the ischium.
Sciatic hernias can exit through the greater or lesser sciatic
hole. The hernial protrusion is located under the gluteus maximus
muscle, sometimes coming out from under its lower edge. Hernial protrusion
is in close contact with the sciatic nerve, so pain may
radiate along the nerve. Sciatic hernias are more often observed in
women. The contents of the hernia can be the small intestine or the greater omentum.

Complications of external abdominal hernias

Strangulated hernia is the most common and
dangerous complication requiring immediate
surgical treatment.
The organs released into the hernial sac are subjected to
compression (usually at the level of the neck of the hernial sac
in the hernial orifice).
Infringement of organs in the hernial sac itself
possibly in one of the chambers of the hernial sac, with
the presence of scar cords that compress organs during
their fusion with each other and with the hernial sac
(for irreducible hernias).

According to the mechanism of occurrence they distinguish:

Elastic entrapment occurs when there is a sudden increase in
intra-abdominal pressure during exercise, coughing,
straining. In this case, overstretching of the hernial orifice occurs, in
resulting in the hernial sac coming out larger than usual,
internal organs. Return of the hernial orifice to its former state
the condition leads to strangulation of the contents of the hernia. With elastic strangulation, compression of organs released into the hernial sac
happens outside.
Fecal impaction is more often observed in older people.
Due to the accumulation of large amounts of intestinal contents in
afferent loop of intestine located in the hernial sac occurs
compression of the efferent loop of this intestine, pressure of the hernial orifice on
the contents of the hernia intensify and lead to fecal impaction
elastic joins. This is how a mixed form arises
infringement.
Mixed

Pathological picture

In the strangulated organ, blood and lymph circulation are disrupted,
due to venous stasis, transudation occurs into the intestinal wall,
its lumen and the cavity of the hernial sac (hernial water).
The intestine becomes cyanotic in color, hernial water remains
transparent. Necrotic changes in the intestinal wall begin with
mucous membrane. The greatest damage occurs in the area
strangulation groove at the site of compression of the intestine by strangulation
ring.
Over time, pathomorphological changes progress,
Gangrene of the strangulated intestine occurs. The intestine becomes blue-black in color, and multiple subserous hemorrhages appear.
The intestine is flabby, does not peristalt, and the mesenteric vessels do not pulsate.
Hernia water becomes cloudy, hemorrhagic with feces
smell. The intestinal wall may undergo perforation with the development
fecal phlegmon and peritonitis.
Intestinal strangulation in the hernial sac is a typical example
strangulation intestinal obstruction.

Clinical picture and diagnosis

Clinical manifestations of strangulated hernia depend
from the form of infringement, the infringed organ,
time elapsed since the infringement.
The main symptoms of a strangulated hernia
are pain in the hernia area and irreducibility
previously freely reducible hernia.

Clinical picture and diagnosis

The intensity of pain varies, sharp pain can
cause a state of shock. Local signs
strangulated hernia is a sharp pain
on palpation, compaction, tension of the hernial
protrusions. Symptom of cough shock
negative. With percussion it is determined
dullness in cases where the hernial sac
contains omentum, bladder, hernial water.
If there is intestine in the hernial sac,
containing gas, then tympanic
percussion sound.

Clinical picture and diagnosis

Elastic entrapment. The onset of complications is associated with an increase
intra-abdominal pressure (physical work, cough, defecation). At
Intestinal strangulation is accompanied by signs of intestinal obstruction:
background of constant acute pain in the abdomen, caused by (pressure of blood vessels and
nerves of the mesentery of the strangulated intestine, cramping pain occurs,
associated with increased peristalsis, there is retention of stool and gases,
vomiting is possible. Without urgent surgical treatment, the patient's condition
quickly worsens: swelling, hyperemia of the skin in the area appears
hernial protrusion, phlegmon develops.
Retrograde infringement. More often the small intestine is strangulated retrogradely when
in the hernial sac there are two intestinal loops, and the intermediate
(connecting) loop is located in the abdominal cavity. Infringement is subjected to
mostly a connecting intestinal loop. Necrosis begins earlier in
intestinal loop located in the abdomen above the strangulating ring. In it
while the intestinal loops located in the hernial sac may still be
viable.
Wall pinching occurs in a narrow pinching ring when
only the part of the intestinal wall opposite the line is infringed
mesenteric attachments. It is observed more often in femoral and inguinal hernias,
less often - in the umbilical. Disorder of lymph and blood circulation in the strangulated
area of ​​the intestine leads to the development of destructive changes, necrosis and
intestinal perforation.

Treatment of strangulated hernias

If a hernia is strangulated, emergency surgery is necessary. It is carried out
so that, without cutting the pinching ring, open the hernial
pouch, prevent the strangulated organs from slipping into the abdominal
cavity. The operation is carried out in several stages.
The first stage is layer-by-layer dissection of tissue to the aponeurosis and
exposure of the hernial sac.
The second stage is opening the hernial sac and removing the hernial water.
To prevent strangulated persons from slipping into the abdominal cavity
organs, the surgeon's assistant holds them with gauze
napkins. It is unacceptable to cut the pinching ring before opening it
hernial sac.
The third stage is dissection of the pinching ring under control
vision, so as not to damage the organs soldered to it from the inside.
The fourth stage - determining the viability of the disadvantaged
organs. This is the most critical stage of the operation. Main
the criteria for the viability of the small intestine are restoration
normal intestinal color, preservation of pulsation of mesenteric vessels,
absence of strangulation groove and subserous hematomas,
restoration of peristaltic contractions of the intestine. Undisputed
signs of intestinal non-viability are dark coloration,
dull serous membrane, flabby wall, lack of pulsation
mesenteric vessels and intestinal peristalsis.

Treatment of strangulated hernias

Fifth stage - resection of a non-viable loop
intestines. From the serous layer visible from the side
the boundaries of necrosis are resected at least 30-40 cm
adductor segment of intestine and 10 cm efferent
segment. Bowel resection is performed when it is detected
in its wall there is a strangulation groove, subserous
hematomas, edema, infiltration and hematoma of the mesentery
intestines.
In case of strangulation of a sliding hernia, it is necessary
determine the viability of a part of an organ, not
covered with peritoneum. If necrosis is detected, the blind
intestines perform resection of the right half of the colon
intestines with ileotransverse anastomosis. At
necrosis of the bladder wall requires resection
modified part of the bubble with overlay
epicystostomy.
The sixth stage is plastic surgery of the hernial orifice. When choosing
plastic surgery method should be preferred
the most simple.

Forecast

Postoperative mortality increases as
lengthening the time that has passed since
infringement before surgery, and is in the first 6 hours -
1.1%, within 6 to 24 hours - 2.1%, after 24 hours -
8.2%; after bowel resection, mortality is 16%,
with hernia phlegmon - 24%.

Complications of self-reduced and forcibly reduced strangulated hernias

A patient with spontaneously strangulated
a reduced hernia must be
hospitalized in the surgical department.
Spontaneously reduced previously strangulated
the intestine can become a source of peritonitis or
intraintestinal bleeding.

Irreversibility

Caused by the presence of adhesions in the hernial sac
internal organs between each other and with the hernial sac,
formed as a result of their traumatization and aseptic
inflammation.
Irreversibility can be partial when one part
the contents of the hernia are reduced into the abdominal cavity, and the other
remains irreparable. Contributes to the development of irreducibility
long-term wearing of the bandage.
Irreversible most often are the umbilical, femoral and
postoperative hernias. Quite often irreparable
hernias are multidimensional. Due to development
multiple adhesions and chambers in the hernial sac irreducible
a hernia is often complicated by strangulation of organs in one of the chambers
hernial sac or development of adhesive obstruction
intestines.

Coprostasis

Coprostasis is stagnation of feces in the large intestine. This
a complication of a hernia in which the contents of the hernial sac
is the large intestine. Coprostasis develops as a result
disorders of intestinal motor function. Its development
contribute to the irreducibility of the hernia, a sedentary image
life, abundant food. Coprostasis is observed more often in obese people
elderly patients, in men with inguinal hernias, in
women - with umbilicals.
The main symptoms are persistent constipation, pain in
stomach, nausea, rarely vomiting. Hernial protrusion slowly
increases as the colon fills with feces
in masses, it is almost painless, slightly tense,
pasty consistency, symptom of cough impulse
positive. The general condition of the patients is of moderate severity.

Prevention of complications

consists of surgical treatment of all patients
with hernias as planned until they develop
complications. The presence of a hernia is an indication for
operations.

Internal abdominal hernias

Internal abdominal hernias are called displacement of organs
abdominal cavity into pockets, crevices and openings of the parietal
peritoneum or into the chest cavity (diaphragmatic hernia). IN
embryonic period of development as a result of the rotation of the primary
the intestine around the axis of the superior mesenteric artery forms the upper
duodenal recess (recessus duodenalis superior - pocket
Treitz). This depression can become a hernial orifice with
formation of an internal strangulated hernia.
Hernia of the lower duodenal recess (recessus duodenalis inferior)
called mesenteric hernias. Loops of small intestine from this
depressions can penetrate between the plates of the mesentery of the colon
intestines right and left.
Most often, the hernial orifices of internal hernias are pockets
peritoneum at the point where the ileum enters the cecum (recessus
ileocaecalis superior et inferior, recessus retrocecalis) or in the area
mesentery of the sigmoid colon (recessus intersigmoideus).
The reasons for the formation of a hernial orifice may be unsutured
during the operation of the gap in the mesentery, greater omentum.
Symptoms of the disease are the same as for acute obstruction
intestines, for which patients are operated on.

Treatment of internal hernias

Apply general principles for the treatment of acute
intestinal obstruction. During surgery
carefully examine the walls of the hernial orifice,
touch determines the absence of pulsation of the large
vessel (superior or inferior mesenteric artery).
The hernial orifice is dissected into avascular
areas. After careful release and
movement of intestinal loops from the hernial sac
he is being sutured.
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