Download presentation on the topic of traumatic brain injury. Presentation on the topic: Damage to the skull and brain

Traumatic brain injury is damage
mechanical energy
skull and intracranial
contents (head
brain, meninges,
vessels, cranial nerves).

Cranial
damage
are the most common among
all types of injuries (45%).
Among the causes of traumatic brain injuries
the first places are occupied by household and
road traffic injuries.
Traumatic brain injury as a cause
death comes first
people aged 20 to 40 years,
so the problem is not only
medical, but also social.

PATHOGENESIS OF CRANIOBRAIN INJURY

PATHOGENESIS OF CRANIAL INJURY
Background
The term "concussion"
first given by Hippocrates.
I.Pti in 1774 allocated
traumatic brain injury three
main forms: concussion,
bruise and compression (commotio,
contusio, compressio).

I.Pti
put forward a theory
"molecular vibrations".
hesitation"
He believed that the injury
causes vibration of the nerves
elements, molecular
changes in brain cells,
which determines
disruption of its function in
in general.

E. Bergmann (1880)
assumed that
in case of brain injury, the damaging force
concentrated at the junction of the trunk and
hemispheres.
He compared the brain to a mushroom,
in which, due to injury, the
massive cap (large hemispheres
brain), and a thin leg (oblong
brain) undergoes flexion and
twisting.
twisting

Modern representations

1.Pressure gradient theory
When injured, the brain becomes
acceleration, which leads to
emergence of high
pressure on the impact side.
At the opposite pole
low
(negative) pressure.
pressure

IN
brain tissue,
located in the area
negative pressure,
cavities are formed and
gas bubbles of various types
magnitude (cavitation).
Negative pressure
lasts very short
time (within
milliseconds), gas
bubbles burst
are torn that
causes damage
capillaries and brain tissue.
brain

2. Rotation theory
In an experiment on monkeys whose arch
the skull has been replaced with a transparent one
material, when used
high-speed filming installed,
that severe head trauma leads to
complex rotational movements
brain simultaneously in 2 - 3 planes
(horizontal, sagittal,
vertical).
Rotational movements are mainly
appear in the cerebral hemispheres, and
fixed stem sections
are injured as a result of their
twisting.
twisting

Pathogenetic mechanisms of all types
Traumatic brain injuries are divided into three
main groups.
1. Acceleration injury (diffuse) occurs when the head hits a large
mass, wide plane or this mass
hits the skull with different
speed. Skull and its contents
get acceleration. Damage
occurs predominantly on
opposite side (type
anti-shock).

2. Impression (local) injury -
when struck by a small object
(stone, stick, hammer, etc.) on the head.
The skull due to its elasticity
bends on impact
hit then pressed
the area is straightened. The bone in this case
may crack and form a linear
fracture of the cranial vault.
skulls
A negative occurs under the impact site.
pressure, causing a lesion to appear
brain damage.
brain
More severe local trauma leads to
depressed fracture of the skull bones.
skulls

3. Compression injury
occurs during the passage
fire projectile through
scull. At the same time, intracranial
pressure can reach 20 – 40
atmospheres, which leads to
destruction of brain tissue and
cranium.

MODERN CLASSIFICATION OF TRANO BRAIN INJURY

Traumatic brain injury is divided into
into two main types - closed and
open.
open
To a closed cranial
injury should be considered
damage in which
no integrity violations
skin and aponeurosis of the fornix
skulls

Open traumatic brain injury
constitute damage in which
there are injuries to the soft tissues of the head,
including aponeurosis.
aponeurosis
Fractures of the base of the skull,
accompanied by leakage of cerebrospinal fluid
from the nose or ear,
ear evidence
violation of the tightness of the cranial
boxes and also refer to open
damage.
damage

In the Russian Federation it is used
classification of closed craniocerebral injury, according to which
The following forms are distinguished:
1) concussion;
2) brain contusion mild, moderate and
severe degrees;
3) diffuse axonal damage
brain;
4) compression of the brain against the background
bruise and without accompanying bruise.

Brain concussion
(commotio cerebri) - most
easy and most common type
closed cranial
injuries. Patients with concussion
brain cases account for 7580% of all hospitalized patients.

By
electron microscopy data
This type of injury is characterized only
minor changes
cell membranes and intercellular
nerve cell contacts.
Concussion is not divided
to degree and is functional,
reversible nerve damage
systems.
systems

Concussion Clinic

There are three leading syndromes:
cerebral, autonomic and
neurotic.
neurotic
The cerebral syndrome manifests itself
impairment of consciousness (stupefaction)
or short-term loss of
a few seconds), headaches,
dizziness, vomiting.

Neurotic syndrome
manifested by general weakness,
apathy, drowsiness,
disturbances in sleep, appetite,
irritability, sometimes
euphoria, decreased criticism towards
to your condition.

Vegetative syndrome
manifested by hyperhidrosis of the palms,
pallor or hyperemia of the skin
covers, violation
dermographism.
There is instability
blood pressure,
pulse lability, seizures
chills, fever.

In
time spent in hospital
patients must undergo
vegetative tests (once every 2-3 days). This
is done to objectify the diagnosis and
identifying the dynamics of pathological
process.
An orthostatic test is used to measure the patient's pulse rate in
horizontal position and then standing. IN
Normally, heart rate should not increase
exceed 20 beats per minute.

Regression
cerebral symptoms and
normalization of vegetative tests
indicate clinical
healing a concussion
brain
Duration of clinical
manifestations usually do not exceed 57 days.
days

Brain contusions
(Contusio cerebri)
differ
predominance
irreversible
morphological
changes in
region
concussion
foci.
outbreaks

Subarachnoid
hemorrhage is always
accompanied by a bruise
brain, because
inevitable
vascular damage
soft cerebral
shells in the outbreak
concussion leads to
blood getting into
cerebrospinal fluid

Mild brain contusions

Focal symptoms
caused by damage to the cortical
parts of one hemisphere of the brain
brain
Light motor movements are noted
violations in the form of asymmetry
reflexes, pathological foot
signs on one side.

Due to
subarachnoid
cerebral hemorrhages, vegetative
and neurotic disorders more
expressed
more pronounced than with a concussion
brain
Meningeal syndrome is associated:
neck muscle stiffness, Kernig's symptoms,
Brudzinsky, photophobia, pain when
movements of the eyeballs.
Duration of clinical manifestations
usually 2-3 weeks.

Moderate brain contusions

Characterized by
emergence
foci of damage to the basal
parts of the cerebral hemispheres and
convexital surface
brain.
brain
This type of brain injury
diagnosed in 100% of patients with
fractures of the base of the skull.

Clinic:
Prolonged loss of consciousness (from
a few seconds to 1-2 hours).
General cerebral symptoms are expressed.
symptoms
Psychomotor impairment may occur
excitement, euphoria, disturbances
psyche.
Rough focal symptoms.
symptoms When
damage to the central gyrus motor and sensory
hemitype disorders.

IN
cases of base fractures
lesions occur on the skull
cranial nerves,
nerves the most
often VIII, VII, II, III, VI nerves.
nerves
Duration of clinical
manifestations – 3-6 weeks.
weeks
Persistent patchy lesions may remain
symptoms of nervous system damage
systems, which leads to
disability of patients.

Severe brain contusions

Characterized by
the occurrence of foci of contusion does not
only the cortex and basal regions
brain, but more
degree of damage
brain stem
brain and diencephalic
areas.

Clinic:
Since a serious injury
the victims are in a comatose state
condition. Duration of loss
consciousness can be from several
days to several weeks, months.
Breathing problems occur immediately
central character,
character to which
quickly join
peripheral respiratory
disorders.
disorders

Rough vegetative,
oculomotor and
bulbar disorders.
Tetraparesis, changes
muscle tone,
tone
double sided
pathological reflexes.

Diffuse axonal brain injury

More often
occurs in children and
teenagers
Pathomorphological
changes - tension and
axonal rupture in white
substance of the hemispheres and trunk
brain
brain

Clinic:
prolonged coma
state,
rough promotion
muscle tone
(hormetonia),
autonomic disorders.

Fractures of the skull bones

Brain contusions in
20 - 35% of cases
accompanied
fractures of the arch bones and
base of the skull.

Fractures of the bones of the cranial vault are:
open (damaged soft tissues
in the area of ​​bone fracture);
closed (soft tissues are not
damaged);
penetrating (with damage
dura mater);
non-penetrating (dura mater)
the shell remains intact).

Linear
fractures are the most
common species
damage
skull bones.

Depressed
there are fractures
impressive
(A) funnel-shaped
impression
fragments,
depressive
(B) - uniform
pressing everything in
fragment.

Clinical manifestations of skull base fractures

Anterior fracture
cranial fossa
Symptom of "glasses" -
hemorrhage in
paraorbital
fiber,
manifesting
after a few
hours or days after
injuries.

Nasal
liquorrhea -
leakage of cerebrospinal fluid
from the nose.
For detection
cerebrospinal fluid impurities in
bloody
liquids
used
symptom
"blurring
spots" on
gauze napkin

Middle cranial fossa fracture:
bleeding
and liquorrhea from the ear;
fall out on the fractured side
functions of the vestibulocochlear and
facial nerves (deafness, paresis
facial muscles);
hemorrhage under the temporal
muscle.

Fracture
rear
cranial
pits:
Hematoma under
aponeurosis
behind
mastoid
process.

Brain compression
May be due to:
1. Intracranial hematoma
(epidural, subdural,
intracerebral, intraventricular).
intraventricular
2. Depressed fracture of the arch bones
skulls
3. Contusion focus,
focal point causing
swelling and displacement of the brain.
4. Subdural hydroma.
hydraulic fluid

Intracranial hematomas

Epidural
hematoma is
limited
accumulation of blood
between the outer
surface
dura mater
shells and
bones of the skull.

Source
emergence
epidural
hematoma is
damaged
branch
shell
arteries.
More often
is happening
average gap
shell
arteries.

Subdural
hematoma is
accumulation of blood
under solid
cerebral
shell.
It arises
most often, when
damage
veins coming from
surfaces
brain to
venous
sinuses.

Intracerebral
I'm a hematoma
formed when
damage
vessels in
areas of injury and
crushing
brain
brain

Clinic:
At the beginning
is happening
compensation
compression of the brain behind
repression account
cerebrospinal fluid from the ventricles
and subarachnoid
fissures of the head
brain
It shows up
asymptomatic
period after
injuries - yes
called
"light
between"

Further
promotion
intracranial
pressure causes
bias
(dislocation)
brain under
crescent
process, in
tenderloin
cerebellar
tentorium, in
occipital
hole.

Brain dislocation
manifests itself:
paresis of the limbs
(mono, or
hemiparesis) to
opposite from
side hematomas;
pupil dilation
on the side of the hematoma;
bradycardia;
epileptic
seizures.

At
the victim has
combinations of any three of
listed signs
(for example, “light
gap", bradycardia,
focal epileptic
seizure)
seizure probability of diagnosis
intracranial hematoma
reaches 90%.

Depressed fractures of the calvarial bones

Clinic:
General cerebral
symptoms characteristic
for brain contusion.
Symptoms
relevant
focal
brain damage
with deep
introduction of fragments

Contusion lesion of the brain

Big
plot
brain destruction
substances,
imbibed
blood, causes swelling
and brain dislocation.
The clinic is
similar to
symptoms
intracranial
hematomas.
hematomas

Acute subdural hydroma

It's limited
accumulation of cerebrospinal fluid in
subdural
space.
space
Clinical picture
the same as with
intracranial
hematoma.

Diagnostic methods

Clinical examination:
History (mechanism of injury,
duration of loss of consciousness,
presence of a “light gap”)
Objective examination (damage
soft tissues of the head, skull bones and
etc.)
Neurological examination

Quantitative assessment of disturbances of consciousness (Glasgow Coma Scale)

Opening Ball
ly
eye
eye
Speech
Ball
ly
Movements
Ball
ly
Spontaneous
speech
5
Localization of pain
irritations
6
5
Movements on command
Spontaneous
opening
eye
4
Separate
phrases
4
Withdrawal
limbs for pain
4
Opening
to the sound
3
Separate
words
3
Pathological
flexion movements
3
Opening
for pain
2
Unintelligible
mumbling
2
Pathological
extensor
movement
2
Absence
reactions
1
Absence
speeches
1
Absence
motor reactions
1

Assessment of brain injury severity using the Glasgow Coma Scale

3-7 points - severe cranial
brain injury.
8-12 points – moderate
traumatic brain injury.
13 -15 points - mild traumatic brain injury.

Radiography
skulls
(craniography)
executed after
examination of the patient
Fracture detection
skull bones
(linear,
depressed) is
reliable sign
brain contusion.

Computed tomography is the primary method for diagnosing traumatic brain injury

At
description of the fracture on CT is necessary
pay attention to: location of the fracture, type
fracture (depressed, linear, comminuted and
etc.), with a depressed fracture - degree
impressions (depression of fragment), direction
fracture plane (for example: fracture
occipital bone with transition to the base
skull through the foramen magnum),
presence of fragments and their localization.

Fracture of the parietal bone on the left side in
two places (with some impression
fragments).

1 - parenchymal intracerebral hematoma of traumatic
character, 2 – area of ​​edema of the brain substance. There is also
subarachnoid hemorrhage - note the right
Sylvian fissure - it is filled with hyperdense content. On
the image in the center and on the right is not visualized quite typically
apparent epidural hematoma (this patient has a fracture
temporal and parietal bones on the right).

Multiple foci of contusion by type
intracerebral hematoma in both
hemispheres of the brain (highlighted
circles),

Depressed
impressive
fracture of the temporal bone.

Lumbar puncture and
cerebrospinal fluid examination.
Allows you to install
Availability
subarachnoid
hemorrhages.

By
the value of cerebrospinal fluid pressure can be
judge liquor hypotension
(pressure below 100 mm water column) or
liquor hypertension (pressure
over 200 mm water column).
If you suspect intracranial
hematoma from lumbar puncture
should abstain.

Echoencephaloscopy

Median echo (M -
echo) – reflected
signal is generated
from the epiphysis, III
ventricle
Direction and
the degree of Mach's displacement indicates
side and magnitude
volumetric process

CT scan. Magnetic resonance imaging

CT scan. Magnetic resonance imaging
For acute
subdural
hematoma
is revealed
sickle-shaped
zone
homogeneous
promotion
density.

Carotid
angiography
For hematomas
typical
identification
avascular
zones.

Overlaying diagnostic milling holes

Produced
at
suspicion of
intracranial
hematoma and
impossibility
carrying out
instrumental
research.
Milling hole
imposed first
only in the anterior regions
temporal bone.
bones

Through
milling
hole is drawn
audit
epidural and
subdural
space
When found
hematomas
carried out
craniotomy and
removal of hematoma.

TREATMENT OF TRANO BRAIN INJURY

All
patients with traumatic brain injury
are subject to hospitalization in medical
institutions.
Patients with concussions and bruises
brain of all degrees are treated
conservatively.
conservatively
Cases of brain compression
require emergency surgery
interventions.
interventions

Conservative treatment of mild traumatic brain injury

Bed rest
Taking medications
aimed at eliminating
cerebral, focal and
autonomic disorders,
normalization of sleep (analgesics,
antihistamines,
sleeping pills).

Conservative treatment of moderate traumatic brain injury

Treatments added:
Neurovegetative blockade
lytic mixtures (droperidol,
aminazine, diphenhydramine).
Restoring cerebral
microcirculation (cavinton,
aminophylline).

At
liquor hypertension –
dehydration with saluretics. At
CSF hypotension - drinking plenty of fluids.
Anti-inflammatory therapy - with
liquor leak.
Regenerative metabolic
therapy (nootropics, cerebrolysin)
Repeated sanitizing lumbar
punctures.

Conservative treatment of severe traumatic brain injury

At the prehospital stage -
first
the queue needs to be restored
upper airway patency
ways: clean the mouth,
nasopharynx from mucus, saliva, vomit
masses,
using mass
mouth dilator, tongue holder,
aspirator.

IN
cases of serious disorders
breathing must be ensured
ventilation of the lungs by any means
(breathing “mouth to mouth”, “mouth to nose”).
Patients are indicated for urgent intubation
trachea,
trachea and if it is impossible
carrying out - tracheostomy.

In the hospital -
artificial
ventilation of the lungs.
Treatments added:
Barbiturates and sodium hydroxybutyrate are used as antihypoxants.
Permanent neurovegetative blockade
lytic mixtures.
Regular sanitation of the tracheobronchial tree.
tree Parenteral, and
after 5-8 days - enteral nutrition
patients through a tube.

Surgical treatment of intracranial hematomas

Apply
osteoplastic or
resection
craniotomy.
At the final stage
operations are performed
infratemporal
decompression removal of the temporal bone
to the base of the skull.

Surgical removal of depressed calvarial fractures

Removal
plots
bone depressions
cranial vault
made from
milling hole,
superimposed next to
with a fracture.

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The presentation on the topic “Medical care for traumatic brain injuries” can be downloaded absolutely free on our website. Subject of the project: life safety. Colorful slides and illustrations will help you engage your classmates or audience. To view the content, use the player, or if you want to download the report, click on the corresponding text under the player. The presentation contains 15 slide(s).

Presentation slides

Slide 1

First aid for traumatic brain injuries and spinal injuries

Completed by: life safety teacher Savustyanenko Viktor Nikolaevich G. Novocherkassk MBOUSOSH No. 6

Slide 2

CRANIOUS BRAIN INJURY Causes What is happening? How to recognize? What to do? Diagnosis Treatment Procedure for providing first aid to a victim of an accident Procedure for providing first aid to a victim

Slide 3

CRANIOUS BRAIN TRAUMA

Treatment of victims with open and closed injuries to the skull and brain has much in common, since they almost always involve a concussion or bruise of the brain, which requires protective therapy, rest, the use of sedatives, and careful monitoring of patients, starting from the advanced stages of evacuation. Providing first aid consists primarily of preventing blood, cerebrospinal fluid or vomit from entering the respiratory tract, for which the wounded person or his head is turned to the side. An aseptic bandage is applied to the wound. At the stage of first medical aid, if necessary, the bandage is corrected and an antibiotic, tetanus toxoid, is administered intramuscularly.

Slide 4

Concussion, traumatic brain injury

It would seem that there is little threat to our brain, because it is protected like no other organ. It is washed by a special liquid, which not only provides the brain with additional nutrition, but also serves as a kind of shock absorber. The brain is covered by several membranes. After all, it is securely “hidden” in the skull. However, head injuries very often result in serious brain problems for a person. All traumatic brain injuries are divided into open and closed. Open injuries are those that damage the soft tissues of the head (skin, subcutaneous tissue, fascia) and the bones of the skull. Closed injuries are somewhat less dangerous, but still unpleasant. They, in turn, are divided into concussion, contusion and compression. Among all brain injuries, concussion ranks first in frequency. Moreover, according to the observations of traumatologists, it occurs more often in women. Although, perhaps they simply seek professional help more often than men.

Slide 5

A concussion can occur as a result of blows, bruises and sudden movements: acceleration or deceleration, such as a fall. The causes of concussions are usually road traffic accidents, domestic, sports and work-related injuries, as well as injuries received as a result of street fights.

Slide 6

What's happening?

What exactly happens to our brain as a result of a concussion is still difficult for doctors to answer unambiguously. After all, if you examine the injured brain using computed tomography, then practically no organic disorders will be detected. Most likely, as a result of a concussion, certain problems arise with the functioning of nerve cells in the brain. At the same time, their nutrition may deteriorate, a slight displacement of the layers of brain tissue may appear, and the connection between some brain centers may be disrupted. A severe concussion can rupture blood vessels and seriously injure certain areas of the brain. The main danger with traumatic brain injuries is intracranial bleeding, since leaked blood can compress and permeate brain structures, disrupting their function and viability. In addition, injury can lead to another serious complication - cerebral edema. Particularly severe are brain injuries complicated by shock and injuries affecting the brain stem, where breathing and blood pressure are regulated.

Slide 7

How to recognize?

After an injury, a person often loses consciousness. This can last from a few seconds to several minutes. The time spent in this state may be one indicator of the severity of the concussion. The extreme degree of loss of consciousness is coma. When a concussion occurs, a person often does not understand where he is, what happened, and has difficulty recognizing the people around him. Another important indicator by which one can judge the severity of brain damage is memory loss: whether a person remembers the moment of injury, and if not, how much of the time before the injury has disappeared from his memory. The greater the memory loss, the more serious the injury. When the victim comes to, he may feel sick and vomit. He often turns pale, feels dizzy and has a headache, there is noise in his ears, it is difficult for him to focus his eyes, his breathing becomes rapid, and his pulse jumps. In the first hours after a concussion, the victim’s pupils are dilated or constricted - a traumatic brain injury of any severity leads to disruption of the nerve pathways responsible for the functioning of the eyes. Surely in the movies you have seen more than once how, when examining an unconscious person, a doctor directs a flashlight beam into the victim’s eyes. This is done to determine the reaction of the pupils. With a mild concussion, the pupils react to light, but sluggishly, and with a severe concussion, there is no reaction at all. In this case, the dilation of only one of the pupils and the lack of reaction in the second is a formidable symptom and may indicate severe damage to one of the hemispheres of the brain.

Slide 8

What to do?

If you suspect a concussion, you must provide first aid to the victim. First, you need to provide the person with complete peace, put him on the bed in a quiet, darkened room. It is better to raise your head slightly. It is very useful to apply cold compresses to the head. Drinking a lot if you have a concussion is not recommended. If the victim is thirsty, make him sweet tea. Alcohol is strictly contraindicated for him! And, of course, be sure to call a doctor, since it is possible that the brain damage is more severe than it seems at first glance. If the patient is in shock, carefully monitor his breathing and blood pressure before the ambulance arrives. In emergency cases, begin artificial respiration and chest compressions.

Slide 9

If you have a concussion, you should consult a traumatologist. He will examine and interview the patient, check reflexes, prescribe an X-ray of the skull, and if more complex brain damage is suspected, refer him for a consultation with a neurologist. There, the patient will undergo a full-scale examination: electroencephalography (EEG), echoencephalography, computed tomography or magnetic resonance imaging of the brain, Dopplerography of cerebral vessels, and spinal puncture. An MRI of the spine may be needed to rule out spinal problems.

Slide 10

Patients with a concussion should remain in bed for at least several days. However, you cannot read, listen to loud music or watch TV. It is necessary to follow all the doctor’s instructions and carefully take the medications prescribed by him. In case of a concussion, the general condition of the victims usually normalizes during the first, less often, the second week after the injury. It must be remembered that a person who has suffered even a mild concussion may develop post-traumatic neurosis or other more serious complications, such as epilepsy. Therefore, some time after recovery, you should definitely visit a neurologist and undergo electroencephalography. Treatment for more serious traumatic brain injuries depends on their severity. In some complicated cases, the help of neurosurgeons may be required.

Slide 11

Procedure for providing first aid to a victim of an accident

In most cases, road traffic accidents occur far from medical facilities and you have to wait a long time for an ambulance. It is this circumstance, as well as the fact that drivers can be useful to victims before others, that obliges them to be able to provide first aid, that is, carry out the simplest emergency measures to save the lives of victims. Drivers of cars and other vehicles, both those involved and not involved in a traffic accident, but who are nearby, are obliged to immediately stop and provide assistance to those in need. Failure to provide assistance to a person in a life-threatening condition is punishable by law. The sequence of actions when providing assistance to victims should be as follows.

Slide 12

Procedure for providing first aid to the victim

1. Organizing an ambulance call. 2. Extracting victims from a broken car. Traffic accidents on the roads are often accompanied by complex fractures, traumatic brain injuries, and spinal injuries. The victim may have several injuries at once. Therefore, you should take it out of the car very carefully. You cannot pull or bend his torso, arms or legs, or pull them out by force. We must first try to eliminate everything that holds the victim back. If a person has lost consciousness and is in an unnatural position, two or three people need to carry him out of the car, trying not to change this position. Special attention should be paid to victims if a spinal fracture is suspected; do not move them unless absolutely necessary, as this can cause paralysis. Such a person must be placed on his back or stomach in such a way that the injury site is not infringed. 3. Providing first aid. When removed from the car, you need to loosen your tie, unfasten your collar and belt so that breathing does not become difficult. For fractures and dislocations of the limbs, it is necessary in all cases to apply splints, and in their absence, fix them with improvised objects (boards, sticks); If the victim experiences bleeding, measures should be taken to temporarily stop it. First aid should be provided quickly and not cause unnecessary pain to the victim. 4. Transportation of victims to a medical facility. When everything possible to save the victims at the scene has been done, but an ambulance cannot be called or it is clear that it will arrive late, care must be taken to transport the victims to the nearest medical facility. You must act with the same caution and attention as when removing them from an emergency vehicle.

Slide 13

When it becomes necessary to lift the victim, you should use the following methods: kneel on the side of the victim, place your hands under the shoulder blade, head, neck and lift him; kneel at the head of the victim, place your hands under the shoulders and lift him up. Under no circumstances is a victim allowed to move independently if the lower extremities, skull, or thoracic or abdominal organs are damaged. If it is necessary to transfer the victim on a stretcher, he is placed carefully, without shaking and in a position comfortable for him. The stretcher is placed next to the victim on the side of the injury. Two people stand next to the patient on one knee, one of them puts his hands under the head, neck and back, the other - under the sacrum and legs. A third person moves a stretcher under the victim. You need to lift the stretcher carefully and at the same time, be sure to walk in step, in short steps, slightly bending your knees. The person walking in front is obliged to warn the person behind about all obstacles on the road. When climbing uphill, the victim is carried head first, when descending from the mountain - feet first, with the exception of cases of damage to the lower extremities. You should always try to keep the victim horizontal. Transportation, depending on the nature of the injury, is carried out according to the following rules: in case of fractures of the skull bones, injuries to the head and brain, in case of fractures of the spine and pelvic bones, the victim is transported only in a horizontal position; for fractures of the ribs and collarbones, transportation in a sitting position is most painless, but when the victim cannot sit, transportation is carried out on a stretcher, giving him a semi-sitting position; for chest injuries, the victim is placed on the wounded side or on the back in a semi-sitting position; when the neck is wounded from the front, the victim is given a semi-sitting position with the head tilted to the chest in the direction of the wound; in case of abdominal wounds and internal bleeding, the victim is placed on his back, a pillow or other object replacing it is placed under the knees and sacrum; in case of fainting, the victim is placed so that his head is lower than his legs.

Slide 14

It is necessary to provide for all the features of the upcoming transportation: its distance and quality of the road, frost and bad weather, the nature of the injuries received, the condition of the victim; take care that it does not deteriorate as a result of transportation. In all cases, take measures to prevent and combat traumatic shock. If there is a large hospital or clinic relatively nearby, it is better to take the victim directly there, bypassing even the nearest medical center. If it is far from a large medical facility, the wounded person should be taken to the nearest medical facility. Upon arrival, do not take him out of the car, but ask medical workers to approach the victim to examine him and decide on further actions. Never leave the wounded unaided and send them without an accompanying person, who may be required to provide the necessary assistance along the way. In addition, through his behavior and conversations, he must strengthen the victim’s confidence in the successful outcome of the incident.

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  • A brain contusion is a traumatic brain injury in which brain tissue is damaged and is characterized by the presence of a focus of necrosis of nervous tissue. Damage after injury can be unilateral or bilateral. The most common sites of damage are in the occipital, temporal and frontal lobes of the brain. Classification of traumatic brain injury: concussion – 80 – 90%; brain contusion – 5 – 12%; compression of the brain – 3 – 5%.


    Causes of occurrence Injury can be obtained as a result of a road traffic accident, injury at work and at home, beatings and beatings, falling from a height, often falling from balconies and windows while intoxicated, during an epileptic attack, diving, falling on the head of heavy objects, during rubble in mines, caves, military operations. Brain damage due to a fracture of the cranial vault.


    Symptoms of a brain contusion In general, symptoms that depend on the severity of the injury include the following: loss of consciousness; headache, dizziness; retrograde amnesia; lack of coordination; nausea and vomiting; changes in consciousness; dysfunction of vision, speech and hearing; dilated pupils; violation of the swallowing reflex; low heart rate; weak, rare breathing; increased blood pressure; loss of sensation in body parts; loss of control over bowel movements and urination; bloody discharge from the nose and ears; paralysis; coma.


    Degrees of brain contusion The severity of brain contusion is determined by the severity of certain clinical manifestations of this condition. A mild degree of brain contusion is characterized by the presence of symptoms that are similar to those of a concussion. However, they are more pronounced. As a rule, in all cases, complete recovery occurs. The average degree of injury is manifested by loss of consciousness, which can vary in duration. Vomiting, headache, and changes in the respiratory and cardiovascular systems appear. The greatest danger to health and life is a severe degree of brain contusion.


    Severe brain contusion Severe brain contusion is a life-threatening condition. It is characterized by a comatose state that lasts several hours; the person may be in a state of psychomotor agitation, which is abruptly replaced by a decadent mood. Depending on the location of the lesion in the brain tissue, focal symptoms appear, which are characterized by disruption of swallowing processes, the functioning of the respiratory and cardiovascular systems, convulsive syndrome, etc.


    First aid for a head injury You must call an ambulance. The victim should be helped to lie on his side to prevent vomit from entering the respiratory tract. It is better to raise your head slightly. It is very useful to apply cold compresses to the head. Drinking a lot if you have a concussion is not recommended. If the victim is thirsty, make him sweet tea. In case of vomiting, it is necessary to empty the oral cavity of vomit, help rinse the mouth, and provide fresh air access to the room. In case of injuries to the soft tissues of the skull, it is necessary to apply a sterile bandage. Sometimes, with minor head injuries, small arteries are damaged, which can lead to massive blood loss. In this case, it is necessary to stop the bleeding. This can usually be done well by pressing the skin against the skull with your fingers in the area of ​​the bleeding vessel, after which a tight sterile bandage with a roller should be applied to this place. In some cases, the cervical spine is immobilized with a rigid collar or improvised material. This is due to the fact that head injuries can often be combined with damage to the cervical spine. For intense headaches, analgesics are used: up to 4 ml of a 50% solution of metamizole sodium intramuscularly or intravenously, 2 ml of ketorolac (30 mg in 1 ml) intramuscularly, etc. Giving tablet forms of analgesics is acceptable in the absence of nausea and vomiting. It is not recommended to use narcotic analgesics for pain relief, as they can depress breathing. The use of analgesics is unacceptable in the presence of concomitant abdominal trauma (it complicates diagnosis); it is inappropriate in patients with deep depression of consciousness. For vomiting and severe nausea, administer 2 ml of metoclopramide solution intramuscularly. Its use is unjustified in case of severe injury, since it depresses the respiratory center. As an antiemetic, you can use 2 ml of a 2% solution of platyphylline hydrotartrate intramuscularly. If possible, the victim is given oxygen inhalations, which prevents oxygen starvation of the brain and its swelling. And, of course, you should definitely call a doctor, since it is possible that the brain damage is more severe than it seems at first glance.



    Prevention and protection measures. There is no specific prevention. In a broader sense, the prevention of traumatic brain injury is: In case of an accident, compliance with traffic and safety rules. In case of sports injury, this is training athletes to fall, wearing protective equipment, training (in boxing and hand-to-hand combat) methods of passive and active protection from blows. In the event of a work injury, this means following safety regulations.

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    Slide 2

    Traumatic brain injury is damage caused by mechanical energy to the skull and intracranial contents (brain, meninges, blood vessels, cranial nerves).

    Slide 3

    Traumatic brain injuries are the most common among all types of injuries (45%). Among the causes of traumatic brain injuries, the first places are occupied by domestic and road traffic injuries. Traumatic brain injury, as a cause of death, is in first place among people aged 20 to 40 years, so the problem is not only medical, but also social.

    Slide 4

    PATHOGENESIS OF CRANIOBRAIN INJURY

    History of the issue The term “concussion” was first introduced by Hippocrates. I. Petit in 1774 identified three main forms for traumatic brain injury: concussion, bruise and compression (commotio, contusio, compressio).

    Slide 5

    I. Petit put forward the theory of “molecular vibrations”. He believed that trauma causes vibration of nerve elements and molecular changes in brain cells, which causes disruption of its function as a whole.

    Slide 6

    E. Bergmann (1880) assumed that in case of brain injury, the damaging force is concentrated at the junction of the brainstem and hemispheres. He compared the brain to a mushroom, in which, when injured, the massive cap (the cerebral hemispheres) is displaced, and the thin leg (the medulla oblongata) is bent and twisted.

    Slide 7

    Modern representations

    1. Pressure gradient theory When an injury occurs, the brain accelerates, which leads to high pressure on the side of the impact. At the opposite pole, low (negative) pressure occurs.

    Slide 8

    In the brain tissue located in the negative pressure zone, cavities and gas bubbles of various sizes (cavitation) are formed. The negative pressure lasts for a very short time (within a millisecond), gas bubbles burst, resulting in damage to capillaries and brain tissue.

    Slide 9

    2. Rotation theory In an experiment on monkeys in which the cranial vault was replaced with a transparent material, using high-speed filming, it was established that severe head injury leads to complex rotational movements of the brain simultaneously in 2 - 3 planes (horizontal, sagittal, vertical). Rotational movements are mainly manifested in the cerebral hemispheres, and the fixed brainstem sections are injured due to their twisting.

    Slide 10

    The pathogenetic mechanisms of all types of traumatic brain injuries are divided into three main groups. 1. Acceleration injury (diffuse) - occurs when the head hits a large mass, a wide plane, or this mass hits the skull at different speeds. The skull and its contents gain speed. Damage occurs predominantly on the opposite side (like a counter-impact).

    Slide 11

    2. Impression (local) injury - when a small object (stone, stick, hammer, etc.) hits the head. The skull, due to its elasticity, bends upon impact, then the depressed area straightens. In this case, the bone can crack and a linear fracture of the cranial vault is formed. Negative pressure develops under the impact site, causing brain damage to occur. More severe local trauma leads to a depressed fracture of the skull bones.

    Slide 12

    3. Compression injury occurs when a gunshot passes through the skull. In this case, intracranial pressure can reach 20–40 atmospheres, which leads to the destruction of brain tissue and the skull.

    Slide 13

    MODERN CLASSIFICATION OF TRANO BRAIN INJURY

    Traumatic brain injury is divided into two main types - closed and open. Closed craniocerebral trauma should include injuries in which there are no violations of the integrity of the skin and the aponeurosis of the cranial vault.

    Slide 14

    An open traumatic brain injury consists of injuries in which there are injuries to the soft tissues of the head, including the aponeurosis. Fractures of the base of the skull, accompanied by leakage of cerebrospinal fluid from the nose or ear, indicate a violation of the tightness of the skull and also belong to open injuries.

    Slide 15

    In the Russian Federation, a classification of closed craniocerebral injury is used, according to which the following forms are distinguished: concussion; mild, moderate and severe brain contusion; diffuse axonal brain damage; compression of the brain due to a bruise and without an accompanying bruise.

    Slide 16

    Concussion (commotio cerebri) is the mildest and most common type of closed head injury. Patients with concussion account for 75-80% of all hospitalized patients.

    Slide 17

    According to electron microscopy, this type of injury is characterized by only minor changes in cell membranes and intercellular contacts of nerve cells. A concussion is not divided into grades and is a functional, reversible injury to the nervous system.

    Slide 18

    Concussion Clinic

    The leading ones are three syndromes: cerebral, vegetative and neurotic. The cerebral syndrome is manifested by impaired consciousness (stunning or short-term loss for a few seconds), headaches, dizziness, and vomiting.

    Slide 19

    Neurotic syndrome is manifested by general weakness, apathy, drowsiness, sleep disturbance, appetite, irritability, sometimes euphoria, and decreased criticism of one’s condition.

    Slide 20

    Vegetative syndrome is manifested by hyperhidrosis of the palms, pallor or hyperemia of the skin, and impaired dermographism. There is instability of blood pressure, lability of pulse, attacks of chills, fever.

    Slide 21

    During their hospital stay, patients must undergo vegetative tests (once every 2-3 days). This is done to objectify the diagnosis and identify the dynamics of the pathological process. An orthostatic test is used - measuring the patient's pulse rate in a horizontal position and then standing. Normally, the heart rate should not exceed 20 beats per minute.

    Slide 22

    Regression of cerebral symptoms and normalization of autonomic tests indicate clinical cure of concussion. The duration of clinical manifestations usually does not exceed 5-7 days.

    Slide 23

    Brain contusions (Contusio cerebri) are distinguished by the predominance of irreversible morphological changes in the area of ​​concussion foci.

    Slide 24

    Subarachnoid hemorrhage always accompanies a brain contusion, since inevitable damage to the vessels of the pia mater at the site of contusion leads to blood entering the cerebrospinal fluid.

    Slide 25

    Mild brain contusions

    Focal symptoms are caused by damage to the cortical parts of one hemisphere of the brain. Mild motor disturbances are noted in the form of asymmetry of reflexes and pathological foot signs on one side.

    Slide 26

    Due to subarachnoid hemorrhage, cerebral, autonomic and neurotic disorders are more pronounced than with a concussion. Meningeal syndrome is added: rigidity of the neck muscles, Kernig's and Brudzinski's symptoms, photophobia, pain when moving the eyeballs. The duration of clinical manifestations is usually 2-3 weeks.

    Slide 27

    Moderate brain contusions

    They are characterized by the appearance of foci of damage to the basal parts of the cerebral hemispheres and the convexital surface of the brain. This type of brain injury is diagnosed in 100% of patients with basal skull fractures.

    Slide 28

    Clinic: Prolonged loss of consciousness (from a few seconds to 1-2 hours). General cerebral symptoms are expressed. Psychomotor agitation, euphoria, and mental disorders may occur. Rough focal symptoms. When the central gyrus is damaged, motor and sensory disorders of the hemitype occur.

    Slide 29

    In cases of fractures of the base of the skull, lesions of the cranial nerves occur, most often the VIII, VII, II, III, VI nerves. The duration of clinical manifestations is 3-6 weeks. Persistent focal symptoms of damage to the nervous system may remain, which leads to disability of patients.

    Slide 30

    Severe brain contusions

    They are characterized by the occurrence of foci of contusion not only in the cortex and basal parts of the brain, but to a greater extent by damage to the stem parts of the brain and the diencephalic region.

    Slide 31

    Clinic: From the moment of severe injury, the victims are in a comatose state. The duration of loss of consciousness can be from several days to several weeks or months. Central respiratory disorders immediately occur, which are quickly joined by peripheral respiratory disorders.

    Slide 32

    Severe autonomic, oculomotor and bulbar disorders. Tetraparesis, changes in muscle tone, bilateral pathological reflexes.

    Slide 33

    Diffuse axonal brain injury

    More common in children and adolescents. Pathomorphological changes - tension and rupture of axons in the white matter of the hemispheres and the brain stem.

    Slide 34

    Clinic: prolonged coma, severe increase in muscle tone (hormetonia), autonomic disorders.

    Slide 35

    Fractures of the skull bones

    Brain contusions in 20 - 35% of cases are accompanied by fractures of the bones of the vault and base of the skull.

    Slide 36

    Fractures of the bones of the cranial vault are: open (damage to the soft tissue in the area of ​​the bone fracture); closed (soft tissues are not damaged); penetrating (with damage to the dura mater); non-penetrating (dura mater remains intact).

    Slide 37

    Linear fractures are the most common type of injury to the skull bones.

    Slide 38

    Depressed fractures are impression (A) - funnel-shaped depression of fragments, depression (B) - uniform depression of the entire fragment.

    Slide 39

    Clinical manifestations of skull base fractures

    Fracture of the anterior cranial fossa The symptom of “glasses” is hemorrhage into the periorbital tissue, which appears several hours or days after the injury.

    Slide 40

    Nasal liquorrhea is the leakage of cerebrospinal fluid from the nose. To detect the presence of cerebrospinal fluid in bloody fluid, the symptom of a “blurring spot” on a gauze napkin is used.

    Slide 41

    Fracture of the middle cranial fossa: bleeding and liquorrhea from the ear; on the side of the fracture, the functions of the vestibulocochlear and facial nerves are lost (deafness, paresis of facial muscles); hemorrhage under the temporalis muscle.

    Slide 42

    Posterior fossa fracture: Hematoma under the aponeurosis behind the mastoid process.

    Slide 43

    Compression of the brain Can be caused by: Intracranial hematoma (epidural, subdural, intracerebral, intraventricular). Depressed fracture of the bones of the cranial vault. A contusion lesion that causes swelling and displacement of the brain. Subdural hydroma.

    Slide 44

    Intracranial hematomas

    An epidural hematoma is a limited collection of blood between the outer surface of the dura mater and the bones of the skull.

    Slide 45

    The source of an epidural hematoma is a damaged branch of the meningeal arteries. Most often, the middle meningeal artery ruptures.

    Slide 46

    A subdural hematoma is an accumulation of blood under the dura mater. It occurs, most often, when the veins running from the surface of the brain to the venous sinuses are damaged.

    Slide 47

    An intracerebral hematoma is formed when blood vessels are damaged in areas of contusion and crushing of the brain.

    Slide 48

    Clinic: First, compensation for compression of the brain occurs due to the displacement of cerebrospinal fluid from the ventricles and subarachnoid fissures of the brain. This is manifested by an asymptomatic period after injury - the so-called “lucid interval”

    Slide 49

    A further increase in intracranial pressure causes displacement (dislocation) of the brain under the falciform process, into the notch of the cerebellar tentorium, into the foramen magnum.

    Slide 50

    Brain dislocation is manifested by: paresis of the limbs (mono- or hemiparesis) on the side opposite to the hematoma; dilation of the pupil on the side of the hematoma; bradycardia; epileptic seizures.

    Slide 51

    If the victim has a combination of any three of the listed signs (for example, “light gap”, bradycardia, focal epileptic seizure), the probability of diagnosing an intracranial hematoma reaches 90%.

    Slide 52

    Depressed fractures of the calvarial bones

    Clinic: General cerebral symptoms characteristic of brain contusion. Symptoms corresponding to focal brain damage due to deep penetration of fragments

    Slide 53

    Contusion lesion of the brain

    A large area of ​​destruction of the brain substance, imbibed by the blood, causes swelling and dislocation of the brain. The clinical picture is similar to that of an intracranial hematoma.

    Slide 55

    Diagnostic methods

    Clinical examination: History (mechanism of injury, duration of loss of consciousness, presence of a “light gap”) Objective examination (damage to the soft tissues of the head, skull bones, etc.) Neurological examination

    Slide 56

    Quantitative assessment of disturbances of consciousness (Glasgow Coma Scale)

  • Slide 57

    Assessment of brain injury severity using the Glasgow Coma Scale

    3-7 points - severe traumatic brain injury. 8-12 points – moderately severe traumatic brain injury. 13 -15 points - mild traumatic brain injury. Slide 61

    Echoencephaloscopy

    Median echo (M - echo) - the reflected signal is formed from the pineal gland, third ventricle. The direction and degree of displacement of the M-echo indicates the side and magnitude of the volumetric process

    Slide 62

    CT scan. Magnetic resonance imaging

    In acute subdural hematoma, a crescent-shaped zone of homogeneous increase in density is revealed.

    Slide 63

    Carotid angiography Hematomas are characterized by the identification of an avascular zone.

    Slide 64

    Overlaying diagnostic milling holes

    Performed if intracranial hematoma is suspected and instrumental studies are impossible. The burr hole is placed primarily in the anterior parts of the temporal bone.

    Slide 65

    An inspection of the epidural and subdural space is performed through the burr hole. If a hematoma is detected, craniotomy is performed and the hematoma is removed.

    Slide 66

    TREATMENT OF TRANO BRAIN INJURY

    All patients with traumatic brain injury are subject to hospitalization in medical institutions. Patients with concussion and brain contusions of all degrees are treated conservatively. Cases of brain compression require emergency surgery.

    Slide 67

    Conservative treatment of mild traumatic brain injury

    Bed rest Taking medications aimed at eliminating cerebral, focal and autonomic disorders, normalizing sleep (analgesics, antihistamines, sleeping pills).

    Slide 68

    Conservative treatment of moderate traumatic brain injury

    Therapeutic agents are added: Neurovegetative blockade with lytic mixtures (droperidol, aminazine, diphenhydramine). Restoring cerebral microcirculation (Cavinton, aminophylline).

    Slide 69

    For liquor hypertension - dehydration with saluretics. For liquor hypotension, drink plenty of fluids. Anti-inflammatory therapy - for liquorrhea. Restorative metabolic therapy (nootropics, Cerebrolysin) Repeated sanitizing lumbar punctures.

    Slide 70

    Conservative treatment of severe traumatic brain injury

    At the prehospital stage, first of all, it is necessary to restore the patency of the upper respiratory tract: clear the oral cavity, nasopharynx of mucus, saliva, vomit, using a mouth dilator, tongue depressor, aspirator.

    Osteoplastic or resection craniotomy is used. At the final stage of the operation, infratemporal decompression is performed - removal of the temporal bone to the base of the skull.

    Slide 74

    Surgical removal of depressed calvarial fractures

    Removal of areas of depression of the bones of the calvarium is carried out from a burr hole placed next to the fracture.

    View all slides

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